J. Theuer

Inducible NOS inhibition, eicosapentaenoic acid supplementation, and angiotensin II-induced renal damage.

F. Luft, H. Haller, R. Dechend, 2005, Kidney international.

Cytochrome P450-Dependent Eicosapentaenoic Acid Metabolites Are Novel BK Channel Activators

M. Gollasch, F. Luft, H. Haller, 2002, Hypertension.

AT1 Receptor Agonistic Antibodies From Preeclamptic Patients Stimulate NADPH Oxidase

G. Wallukat, R. Brandes, R. Dietz, 2003, Circulation.

Aspirin inhibits NF‐κB and protects from angiotensin II‐induced organ damage

D. Ganten, R. Dietz, F. Luft, 2001 .

NF-κB Inhibition Ameliorates Angiotensin II–Induced Inflammatory Damage in Rats

D. Ganten, F. Luft, H. Haller, 2000 .

Immunosuppressive treatment protects against angiotensin II-induced renal damage.

M. Zenke, F. Luft, H. Haller, 2002, The American journal of pathology.

Angiotensin II (AT(1)) receptor blockade reduces vascular tissue factor in angiotensin II-induced cardiac vasculopathy.

D. Ganten, F. Luft, T. Luther, 2000, The American journal of pathology.

NF-kappaB inhibition ameliorates angiotensin II-induced inflammatory damage in rats.

D. Ganten, F. Luft, H. Haller, 2000, Hypertension.

Aspirin inhibits NF-kappaB and protects from angiotensin II-induced organ damage.

D. Ganten, R. Dietz, F. Luft, 2001, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.

AT(1) receptor agonistic antibodies from preeclamptic patients cause vascular cells to express tissue factor.

G. Wallukat, F. Luft, N. Mackman, 2000, Circulation.