M. Sánchez-Aragó

发表

Sensitivity to anti-Fas is independent of increased cathepsin D activity and adrenodoxin reductase expression occurring in NOS-3 overexpressing HepG2 cells.

J. Muntané, M. L. de la Mata, M. Rodríguez-Perálvarez, 2015, Biochimica et biophysica acta.

Short-term exposure of nontumorigenic human bronchial epithelial cells to carcinogenic chromium(VI) compromises their respiratory capacity and alters their bioenergetic signature

J. Cerveira, J. Cuezva, M. Sánchez-Aragó, 2014, FEBS open bio.

Protease activity of MALT 1 : a mystery unravelled

I. Martínez-Reyes, M. Sánchez-Aragó, 2012 .

Lack of GDAP1 Induces Neuronal Calcium and Mitochondrial Defects in a Knockout Mouse Model of Charcot-Marie-Tooth Neuropathy

Paula Juárez, R. Chrast, J. Cuezva, 2015, PLoS genetics.

The bioenergetic signature of isogenic colon cancer cells predicts the cell death response to treatment with 3-bromopyruvate, iodoacetate or 5-fluorouracil

J. Cuezva, M. Sánchez-Aragó, 2011, Journal of Translational Medicine.

Adalimumab Reduces Photoreceptor Cell Death in A Mouse Model of Retinal Degeneration

E. J. de la Rosa, J. Millán, D. Hervás, 2015, Scientific Reports.

Glucose avidity of carcinomas.

A. Ortega, María Sánchez-Aragó, José M Cuezva, 2009, Cancer letters.

The mitochondrial ATPase inhibitory factor 1 triggers a ROS-mediated retrograde prosurvival and proliferative response.

J. Cuezva, L. Formentini, L. Sánchez-Cenizo, 2012, Molecular cell.

The tumor suppressor function of mitochondria: translation into the clinics.

A. Ortega, J. Cuezva, I. Willers, 2009, Biochimica et biophysica acta.

Outcomes of single versus double hormone receptor-positive breast cancer. A GEICAM/9906 sub-study.

A. Lluch, E. Alba, M. Martín, 2018, European journal of cancer.

A message emerging from development: the repression of mitochondrial β-F1-ATPase expression in cancer

A. Ortega, J. Cuezva, S. Sala, 2007, Journal of bioenergetics and biomembranes.

PKA Phosphorylates the ATPase Inhibitory Factor 1 and Inactivates Its Capacity to Bind and Inhibit the Mitochondrial H(+)-ATP Synthase.

Javier García-Bermúdez, María Sánchez-Aragó, Beatriz Soldevilla, 2015, Cell reports.

AMPK and GCN2-ATF4 signal the repression of mitochondria in colon cancer cells.

I. Martínez-Reyes, J. Cuezva, M. Sánchez-Aragó, 2012, The Biochemical journal.

Mitochondrial bioenergetics and dynamics interplay in complex I-deficient fibroblasts.

B. Merinero, J. Arenas, Miguel Ángel Martín, 2010, Biochimica et biophysica acta.

Selection of cancer cells with repressed mitochondria triggers colon cancer progression.

J. Cuezva, M. Sánchez-Aragó, M. Chamorro, 2010, Carcinogenesis.

Prognostic role for the derived neutrophil-to-lymphocyte ratio in early breast cancer: a GEICAM/9906 substudy

A. Lluch, E. Alba, M. Martín, 2018, Clinical and Translational Oncology.

Mitochondria-mediated energy adaption in cancer: the H(+)-ATP synthase-geared switch of metabolism in human tumors.

J. Cuezva, L. Formentini, M. Sánchez-Aragó, 2013, Antioxidants & redox signaling.

1 Expression , regulation and clinical relevance of the ATPase Inhibitory Factor 1 ( IF 1 ) in human cancers

Á. Juarranz, L. Nájera, I. Martínez-Reyes, 2013 .

Loss of the mitochondrial bioenergetic capacity underlies the glucose avidity of carcinomas.

F. López-Ríos, C. Ballestín, F. Pozo-Rodríguez, 2007, Cancer research.

Hif-1α Knockdown Reduces Glycolytic Metabolism and Induces Cell Death of Human Synovial Fibroblasts Under Normoxic Conditions

J. Pablos, F. Blanco, J. Cañete, 2017, Scientific Reports.

Down-regulation of oxidative phosphorylation in the liver by expression of the ATPase inhibitory factor 1 induces a tumor-promoter metabolic state

Á. Valverde, J. Zapata, J. Satrústegui, 2015, Oncotarget.

Up-regulation of the ATPase Inhibitory Factor 1 (IF1) of the Mitochondrial H+-ATP Synthase in Human Tumors Mediates the Metabolic Shift of Cancer Cells to a Warburg Phenotype*

A. Ortega, P. García-Huerta, J. Cuezva, 2010, The Journal of Biological Chemistry.

Expression, regulation and clinical relevance of the ATPase inhibitory factor 1 in human cancers

Á. Juarranz, L. Nájera, I. Martínez-Reyes, 2013, Oncogenesis.

Quantitative analysis of proteins of metabolism by reverse phase protein microarrays identifies potential biomarkers of rare neuromuscular diseases

Miguel Ángel Martín, J. Cuezva, F. Santacatterina, 2015, Journal of Translational Medicine.

Mitochondrial response to the BCKDK-deficiency: Some clues to understand the positive dietary response in this form of autism.

B. Merinero, M. Ugarte, R. Artuch, 2016, Biochimica et biophysica acta.

Plasma metabolome and skin proteins in Charcot-Marie-Tooth 1A patients

C. Ibáñez, C. Simó, C. Espinós, 2017, PloS one.

Pyruvate kinase M2 and the mitochondrial ATPase Inhibitory Factor 1 provide novel biomarkers of dermatomyositis: a metabolic link to oncogenesis

J. Grau, J. Cuezva, G. Garrabou, 2017, Journal of Translational Medicine.

IF1 reprograms energy metabolism and signals the oncogenic phenotype in cancer

J. Cuezva, L. Formentini, M. Sánchez-Aragó, 2012, Cell cycle.

Degradation of IF1 controls energy metabolism during osteogenic differentiation of stem cells

I. Martínez-Reyes, J. Cuezva, F. Santacatterina, 2013, EMBO reports.

Luminal androgen receptor role and pathological complete response rate to neoadjuvant chemotherapy in triple negative breast cancer

J. M. Jerez, J. Albanell, A. Prat, 2016 .

Quantitative analysis of proteins of metabolism by reverse phase protein microarrays identifies potential biomarkers of rare neuromuscular diseases

Miguel Ángel Martín, J. Cuezva, F. Santacatterina, 2015, Journal of Translational Medicine.

Dataset reporting BCKDK interference in a BCAA-catabolism restricted environment

B. Merinero, M. Ugarte, R. Artuch, 2016, Data in brief.