O. Alsheich-Bartok

发表

E6AP is required for replicative and oncogene-induced senescence in mouse embryo fibroblasts

Y. Haupt, K. Wolyniec, S. Haupt, 2012, Oncogene.

E6ap Ubiquitin Ligase Regulates Pml-induced Senescence in Myc-driven Lymphomagenesis Neoplastic Transformation Requires the Elimination of Key Tumor Suppressors, Which May Result from E3 Ligase-mediated Proteasomal Degradation. We Previously Demonstrated a Key Role for the E3 Ubiqui- Tin Ligase E6ap

P. Pandolfi, S. Fox, S. Lowe, 2022 .

E6AP promotes the degradation of the PML tumor suppressor

M. Scheffner, Y. Haupt, S. Haupt, 2009, Cell Death and Differentiation.

The p53-Mdm2 loop: a critical juncture of stress response.

Y. Haupt, S. Haupt, Zehavit Goldberg, 2014, Sub-cellular biochemistry.

MDM2 and Fbw7 cooperate to induce p63 protein degradation following DNA damage and cell differentiation

E. Shaulian, Y. Haupt, L. Guerrini, 2010, Journal of Cell Science.

Promyelocytic leukemia protein is required for gain of function by mutant p53.

G. Blandino, Y. Haupt, S. Haupt, 2009, Cancer research.

Clues from worms: a Slug at Puma promotes the survival of blood progenitors

Y. Haupt, S. Haupt, O. Alsheich-Bartok, 2006, Cell Death and Differentiation.

PML enhances the regulation of p53 by CK1 in response to DNA damage

E. Appella, Y. Haupt, S. Haupt, 2008, Oncogene.

The Association of MHC Class I Proteins with the 2B4 Receptor Inhibits Self-Killing of Human NK Cells

O. Mandelboim, M. Elboim, Gili Betser-Cohen, 2010, The Journal of Immunology.

Human NK Cells with the 2 B 4 Receptor Inhibits Self-Killing of The Association of MHC Class I Proteins

O. Mandelboim, M. Elboim, Gili Betser-Cohen, 2010 .