Danielle L. Burke

Sustained hypoxia promotes the development of a pulmonary artery-specific chronic inflammatory microenvironment.

A. Anwar, M. Frid, K. Stenmark, 2009, American journal of physiology. Lung cellular and molecular physiology.

Hypoxia-induced pulmonary vascular remodeling requires recruitment of circulating mesenchymal precursors of a monocyte/macrophage lineage.

M. Frid, K. Stenmark, N. Van Rooijen, 2006, The American journal of pathology.

Prostacyclin Inhibits IFN-γ-Stimulated Cytokine Expression by Reduced Recruitment of CBP/p300 to STAT1 in a SOCS-1-Independent Manner1

K. Stenmark, D. Strassheim, M. Geraci, 2009, The Journal of Immunology.

Cytosolic Phospholipase A2 Translocates to Forming Phagosomes during Phagocytosis of Zymosan in Macrophages*[boxs]

C. Leslie, M. Girotti, John H. Evans, 2004, Journal of Biological Chemistry.

Circulating mononuclear cells with a dual, macrophage-fibroblast phenotype contribute robustly to hypoxia-induced pulmonary adventitial remodeling.

M. Frid, K. Stenmark, T. Carpenter, 2005, Chest.

Sustained hypoxia leads to the emergence of cells with enhanced growth, migratory, and promitogenic potentials within the distal pulmonary artery wall.

M. Frid, Min Li, K. Stenmark, 2009, American journal of physiology. Lung cellular and molecular physiology.

Prostacyclin Inhibits IFN-γ-Stimulated Cytokine Expression by Reduced Recruitment of CBP/p300 to STAT1 in a SOCS-1-Independent Manner1

K. Stenmark, D. Strassheim, M. Geraci, 2009, The Journal of Immunology.

Sustained hypoxia leads to the emergence of cells with enhanced growth, migratory, and promitogenic potentials within the distal pulmonary artery wall.

M. Frid, Min Li, K. Stenmark, 2009, American journal of physiology. Lung cellular and molecular physiology.