Yu Hu

发表

Effect of Carcinogenic Acrolein on DNA Repair and Mutagenic Susceptibility*

M. Tang, Xue-Ru Wu, L. Gu, 2012, The Journal of Biological Chemistry.

AFB1 hepatocarcinogenesis is via lipid peroxidation that inhibits DNA repair, sensitizes mutation susceptibility and induces aldehyde-DNA adducts at p53 mutational hotspot codon 249

M. Tang, S. Amin, D. Desai, 2017, Oncotarget.

NFAT3 is specifically required for TNF-α-induced cyclooxygenase-2 (COX-2) expression and transformation of Cl41 cells

Jingxia Li, Chuanshu Huang, Yan Yan, 2006, Journal of Cell Science.

Effect of arsenic on transcription factor AP-1 and NF-kappaB DNA binding activity and related gene expression.

X. Jin, E. T. Snow, Yu Hu, 2002, Toxicology letters.

Regulation of redox and DNA repair genes by arsenic: Low dose protection against oxidative stress?

M. Schuliga, C. Klein, E. T. Snow, 2003 .

Modulation of DNA Repair and Glutathione Levels in Human Keratinocytes by Micromolar Arsenite

E. T. Snow, S. Chouchane, Yu Hu, 1999 .

Acrolein induced DNA damage, mutagenicity and effect on DNA repair.

M. Tang, Wenwei Hu, Zhaohui Feng, 2011, Molecular nutrition & food research.

Acrolein- and 4-Aminobiphenyl-DNA adducts in human bladder mucosa and tumor tissue and their mutagenicity in human urothelial cells

Nicholas M. Donin, H. Lepor, M. Tang, 2014, Oncotarget.

Aldehydes are the predominant forces inducing DNA damage and inhibiting DNA repair in tobacco smoke carcinogenesis

W. Rom, H. Lepor, M. Tang, 2018, Proceedings of the National Academy of Sciences.

DNA damage, DNA repair and carcinogenicity: Tobacco smoke versus electronic cigarette aerosol.

M. Tang, Lung-Chi Chen, Sung-Hyun Park, 2021, Mutation research. Reviews in mutation research.

Alteration of GSH level, gene expression and cell transformation in NIH3T3 cells by chronic exposure to low dose of arsenic

Guoquan Wang, E. T. Snow, Yu Hu, 2003 .

Mutagenicity and sequence specificity of acrolein-DNA adducts.

M. Tang, Hsiang-Tsui Wang, Siyi Zhang, 2009, Chemical research in toxicology.

Acrolein is a major cigarette-related lung cancer agent: Preferential binding at p53 mutational hotspots and inhibition of DNA repair

M. Tang, Wenwei Hu, Zhaohui Feng, 2006, Proceedings of the National Academy of Sciences.

Direct evidence for the critical role of NFAT3 in benzo[a]pyrene diol-epoxide-induced cell transformation through mediation of inflammatory cytokine TNF induction in mouse epidermal Cl41 cells.

Jingxia Li, Chuanshu Huang, Yan Yan, 2007, Carcinogenesis.

Permissive Role of Nitric Oxide in Endothelin-induced Migration of Endothelial Cells*

Ivar Giaever, Charles R. Keese, M. Goligorsky, 1997, The Journal of Biological Chemistry.

PAH-DNA adducts in a Chinese population: relationship to PAH exposure, smoking and polymorphisms of metabolic and DNA repair genes

M. Tang, Donghui Li, X. Xue, 2008, Biomarkers : biochemical indicators of exposure, response, and susceptibility to chemicals.

Acrolein- and 4-Aminobiphenyl-DNA adducts in human bladder mucosa and tumor tissue and their mutagenicity in human urothelial cells

H. Lepor, M. Tang, W. Huang, 2014, OncoTarget.

Sub-toxic arsenite induces a multi-component protective response against oxidative stress in human cells,

M. Schuliga, E. T. Snow, S. Chouchane, 2000 .

Sensitive biomarker of polycyclic aromatic hydrocarbons (PAHs): urinary 1-hydroxyprene glucuronide in relation to smoking and low ambient levels of exposure

M. Tang, X. Xue, X. Huang, 2006, Biomarkers : biochemical indicators of exposure, response, and susceptibility to chemicals.