C. Cave

发表

Early infection during burn-induced inflammatory response results in increased mortality and p38-mediated neutrophil dysfunction.

J. Dattilo, C. Caldwell, J. Tschöp, 2010, American journal of physiology. Regulatory, integrative and comparative physiology.

Interleukin-7 (IL-7) Treatment Accelerates Neutrophil Recruitment through γδ T-Cell IL-17 Production in a Murine Model of Sepsis

Caldwell, R. Hotchkiss, David A Hildeman, 2010, Infection and Immunity.

T-CELL ACTIVATION DIFFERENTIALLY MEDIATES THE HOST RESPONSE TO SEPSIS

J. Dattilo, David A Hildeman, C. Caldwell, 2010, Shock.

SepsisProduction in a Murine Model of T-Cell δ γ Neutrophil Recruitment through Interleukin-7 (IL-7) Treatment Accelerates

R. Hotchkiss, David A Hildeman, Cristina Mazuski, 2014 .

The Cannabinoid Receptor 2 Is Critical for the Host Response to Sepsis

M. Tschöp, A. Lentsch, J. Tschöp, 2009 .

FORMATION OF FOCAL ADHESION-LIKE STRUCTURES IN CIRCULATING HUMAN NEUTROPHILS AFTER SEVERE INJURY: TRIGGERING OF A TISSUE-PHASE RESPONSE IN THE VASCULAR SPACE

K. Umanskiy, Mark A. Williams, J. Solomkin, 2006, Shock.

Chemokine regulation of neutrophil function in surgical inflammation.

J. Solomkin, M. Williams, G. Quaid, 1999, Archives of surgery.

The Cannabinoid Receptor 2 Is Critical for the Host Response to Sepsis1

J. Dattilo, M. Tschöp, A. Lentsch, 2009, The Journal of Immunology.

The Cannabinoid Receptor 2 Is Critical for the Host Response to Sepsis 1

J. Dattilo, M. Tschöp, A. Lentsch, 2009 .

the Host Response to Sepsis The Cannabinoid Receptor 2 Is Critical for

J. Dattilo, M. Tschöp, A. Lentsch, 2009 .

ALTERATIONS IN MEMBRANE CHOLESTEROL CAUSE MOBILIZATION OF LIPID RAFTS FROM SPECIFIC GRANULES AND PRIME HUMAN NEUTROPHILS FOR ENHANCED ADHERENCE-DEPENDENT OXIDANT PRODUCTION

J. Solomkin, A. Lentsch, C. Robinson, 2007, Shock.

CXCR2 regulation of tumor necrosis factor-alpha adherence-dependent peroxide production is significantly diminished after severe injury in human neutrophils.

J. Solomkin, M. Williams, G. Quaid, 2001, The Journal of trauma.

Preferential loss of CXCR-2 receptor expression and function in patients who have undergone trauma.

J. Solomkin, M. Williams, C. Robinson, 1999, Archives of surgery.

Mechanisms of human neutrophil oxidant production after severe injury.

G. Bokoch, J. Solomkin, R. Hennigan, 2001, Surgery.

INTERLEUKIN 8 DIMERIZATION AS A MECHANISM FOR REGULATION OF NEUTROPHIL ADHERENCE-DEPENDENT OXIDANT PRODUCTION

Mark A. Williams, J. Solomkin, A. Lentsch, 2005, Shock.

Mononuclear cell line THP-1 internalizes bactericidal/permeability-increasing protein by a non-receptor-mediated mechanism consistent with pinocytosis.

J. Solomkin, M. N. Marra, R. J. Burnett, 1996, Archives of surgery.

Tumor necrosis factor alpha regulates CXC chemokine receptor expression and function.

M. Lieberman, G. Babcock, J. Solomkin, 1999, Shock.

Divergent adaptive and innate immunological responses are observed in humans following blunt trauma

J. Solomkin, A. Lentsch, C. Caldwell, 2010, BMC Immunology.

NADPH oxidase activation in fibronectin adherent human neutrophils: A potential role for beta1 integrin ligation.

K. Umanskiy, Mark A. Williams, J. Cuschieri, 2003, Surgery.

Regulation of TNF-α Receptor Expression on Human Neutrophils by Various Cell Activating Factors

J. Solomkin, F. Zemlan, C. Cave, 1994 .

Chemokine regulation of neutrophil function in surgical inflammation.

Mark A. Williams, G. Quaid, C. Cave, 1999, Archives of surgery.