M. Salas

发表

Chemical chaperones improve the functional recovery of stunned myocardium by attenuating the endoplasmic reticulum stress

J. I. Mariángelo, L. Vittone, M. Said, 2020, Acta physiologica.

Reversible redox modifications of ryanodine receptor ameliorate ventricular arrhythmias in the ischemic-reperfused heart.

X. Wehrens, P. Donoso, G. Schinella, 2016, American journal of physiology. Heart and circulatory physiology.

CaMKII inhibition protects against necrosis and apoptosis in irreversible ischemia-reperfusion injury.

R. Hajjar, E. Kranias, E. Portiansky, 2007, Cardiovascular research.

Angiotensin II–Induced Oxidative Stress Resets the Ca2+ Dependence of Ca2+–Calmodulin Protein Kinase II and Promotes a Death Pathway Conserved Across Different Species

A. Mattiazzi, J. Palomeque, C. Valverde, 2009, Circulation research.

Contractile recovery from acidosis in toad ventricle is independent of intracellular pH and relies upon Ca2+ influx

M. Vila-Petroff, A. Mattiazzi, R. A. Venosa, 2006, Journal of Experimental Biology.

Subcellular mechanisms of the positive inotropic effect of angiotensin II in cat myocardium

E. Aiello, A. Mattiazzi, M. Petroff, 2000, The Journal of physiology.

The signalling pathway of CaMKII-mediated apoptosis and necrosis in the ischemia/reperfusion injury.

E. Kranias, E. Portiansky, M. Kaetzel, 2010, Journal of molecular and cellular cardiology.

Early ischemic preconditioning protects against infarct and myocardial stunning in isolated rat heart

A. Moreyra, Investigaciones Científicas, S. Mosca, 1999 .

CaMKII-dependent phosphorylation of cardiac ryanodine receptors regulates cell death in cardiac ischemia/reperfusion injury.

X. Wehrens, J. Brown, G. Rinaldi, 2014, Journal of molecular and cellular cardiology.

Positive inotropic and negative lusitropic effect of angiotensin II: intracellular mechanisms and second messengers.

M. Vila-Petroff, A. Mattiazzi, J. Palomeque, 2001, Journal of molecular and cellular cardiology.

Frequency‐dependent acceleration of relaxation in mammalian heart: a property not relying on phospholamban and SERCA2a phosphorylation

A. Mattiazzi, P. Ferrero, J. Palomeque, 2005, The Journal of physiology.

Increased Na+/Ca2+ Exchanger Expression/Activity Constitutes a Point of Inflection in the Progression to Heart Failure of Hypertensive Rats

E. Portiansky, G. Rinaldi, A. Mattiazzi, 2014, PloS one.

CaMKII mediates angiotensin II-induced cardiomyocytes apoptosis: Role of Ca2+, ROS and p38MAPK

A. Mattiazzi, J. Palomeque, C. Valverde, 2008 .