Susanne Wiemann

发表

Transfer of the Experimental Autoimmune Glaucoma Model from Rats to Mice—New Options to Study Glaucoma Disease

S. Joachim, A. Faissner, H. B. Dick, 2019, International journal of molecular sciences.

Optic Nerve Degeneration after Retinal Ischemia/Reperfusion in a Rodent Model

S. Joachim, A. Faissner, G. Stute, 2015, Front. Cell. Neurosci..

Ischemic injury leads to extracellular matrix alterations in retina and optic nerve

S. Joachim, A. Faissner, G. Stute, 2017, Scientific Reports.

Immunomodulatory role of the extracellular matrix protein tenascin-C in neuroinflammation.

A. Faissner, J. Reinhard, Susanne Wiemann, 2019, Biochemical Society transactions.

Loss of the Extracellular Matrix Molecule Tenascin-C Leads to Absence of Reactive Gliosis and Promotes Anti-inflammatory Cytokine Expression in an Autoimmune Glaucoma Mouse Model

S. Joachim, A. Faissner, S. Reinehr, 2020, bioRxiv.

Knock-Out of Tenascin-C Ameliorates Ischemia-Induced Rod-Photoreceptor Degeneration and Retinal Dysfunction

S. Joachim, Natalie Wagner, J. Reinhard, 2021, Frontiers in Neuroscience.

Extracellular Matrix Remodeling in the Retina and Optic Nerve of a Novel Glaucoma Mouse Model

A. Faissner, J. Reinhard, Susanne Wiemann, 2021, Biology.

Early remodelling of the extracellular matrix proteins tenascin‐C and phosphacan in retina and optic nerve of an experimental autoimmune glaucoma model

S. Joachim, A. Faissner, J. Woestmann, 2016, Journal of cellular and molecular medicine.

Heterozygous Meg2 Ablation Causes Intraocular Pressure Elevation and Progressive Glaucomatous Neurodegeneration

G. Downey, S. Joachim, A. Faissner, 2018, Molecular Neurobiology.

Neural extracellular matrix regulates visual sensory motor integration

D. Rathbun, Hamed Shabani, A. Faissner, 2023, bioRxiv.

Heterozygous Meg2 Ablation Causes Intraocular Pressure Elevation and Progressive Glaucomatous Neurodegeneration

G. Downey, S. Joachim, A. Faissner, 2018, Molecular Neurobiology.