W. Seemann

发表

Lack of Gαi2 leads to dilative cardiomyopathy and increased mortality in β1-adrenoceptor overexpressing mice.

K. Mohr, L. Birnbaumer, B. Nürnberg, 2015, Cardiovascular research.

Engineered Context-Sensitive Agonism: Tissue-Selective Drug Signaling through a G Protein-Coupled Receptor

U. Holzgrabe, B. Fleischmann, P. Sasse, 2017, The Journal of Pharmacology and Experimental Therapeutics.

Title : Engineered context-sensitive agonism – tissue-selective drug signaling through a G protein-coupled receptor

D. Wenzel, Theresa Bödefeld, W. Seemann, 2016 .

Pro‐fibrotic processes in human lung fibroblasts are driven by an autocrine/paracrine endothelinergic system

E. Kostenis, K. Mohr, WK Seemann, 2013, British journal of pharmacology.

Agonists with supraphysiological efficacy at the muscarinic M 2 acetylcholine receptor 1 Running title : Supraphysiological M 2 receptor activation

U. Holzgrabe, E. Kostenis, K. Mohr, 2012 .

Rational design of partial agonists for the muscarinic m1 acetylcholine receptor.

U. Holzgrabe, M. Decker, K. Mohr, 2015, Journal of medicinal chemistry.

Applying label-free dynamic mass redistribution technology to frame signaling of G protein–coupled receptors noninvasively in living cells

G. Milligan, E. Kostenis, K. Mohr, 2011, Nature Protocols.

The β2-subtype of adrenoceptors mediates inhibition of pro-fibrotic events in human lung fibroblasts

E. Kostenis, K. Mohr, R. Gosens, 2011, Naunyn-Schmiedeberg's Archives of Pharmacology.

Lack of Gαi2 leads to dilative cardiomyopathy and increased mortality in β1-adrenoceptor overexpressing mice.

K. Mohr, L. Birnbaumer, B. Nürnberg, 2015, Cardiovascular research.

Agonists with supraphysiological efficacy at the muscarinic M2 ACh receptor

U. Holzgrabe, E. Kostenis, K. Mohr, 2013, British journal of pharmacology.