Li Li

Targeting Hypoxia-inducible Factor-1α With Tf-PEI-shRNA Complex via Transferrin Receptor-mediated Endocytosis Inhibits Melanoma Growth.

Changzheng Huang, Y. Tu, Jing Yang, 2009, Molecular therapy : the journal of the American Society of Gene Therapy.

Targeting hypoxia-inducible factor-1alpha with Tf-PEI-shRNA complex via transferrin receptor-mediated endocytosis inhibits melanoma growth.

Changzheng Huang, Y. Tu, Jing Yang, 2009, Molecular therapy : the journal of the American Society of Gene Therapy.

Hypoxia: dual effect on the expression of transferrin receptor in human melanoma A375 cell line

J. Tao, G. Shen, Jing Liu, 2007, Experimental dermatology.

Overexpression of programmed death-1 ligand-1 on NIT cells lead to negative regulation of allogeneic lymphocyte activation.

W. Dai, Jing Yang, G. Shen, 2010, Cellular immunology.

IL-10 Gene Modified Dendritic Cells Inhibit T Helper Type 1-Mediated Alloimmune Responses and Promote Immunological Tolerance in Diabetes

Wei Zhou, P. Lei, G. Shen, 2008, Cellular and Molecular Immunology.

Engineering and characterization of a baculovirus-expressed mouse/human chimeric antibody against transferrin receptor.

W. Xing, P. Lei, G. Hu, 2009, Protein engineering, design & selection : PEDS.

An anti-transferrin receptor antibody enhanced the growth inhibitory effects of chemotherapeutic drugs on human non-hematopoietic tumor cells.

W. Xing, P. Lei, G. Shen, 2008, International immunopharmacology.

Expression of GLUT-1 in psoriasis and the relationship between GLUT-1 upregulation induced by hypoxia and proliferation of keratinocyte growth.

J. Tao, G. Shen, Xue Wen, 2008, Journal of dermatological science.

Glucose regulated proteins 78 protects insulinoma cells (NIT-1) from death induced by streptozotocin, cytokines or cytotoxic T lymphocytes.

P. Lei, G. Shen, P. Wang, 2007, The international journal of biochemistry & cell biology.