Kohji Murakami

DHCR24-knockout embryonic fibroblasts are susceptible to serum withdrawal-induced apoptosis because of dysfunction of caveolae and insulin-Akt-Bad signaling.

H. Seo, F. Kambe, Xia Cao, 2006, Endocrinology.

Structure–activity studies on 1,3-dioxane-2-carboxylic acid derivatives, a novel class of subtype-selective peroxisome proliferator-activated receptor α (PPARα) agonists

Y. Sugiyama, T. Asaki, K. Kuwabara, 2008 .

Discovery of a novel class of 1,3-dioxane-2-carboxylic acid derivatives as subtype-selective peroxisome proliferator-activated receptor alpha (PPARalpha) agonists.

Y. Sugiyama, T. Asaki, K. Kuwabara, 2008, Bioorganic & medicinal chemistry letters.

A comparison of vasodilation mode among selexipag (NS‐304; [2‐{4‐[(5,6‐diphenylpyrazin‐2‐yl)(isopropyl)amino]butoxy}‐N‐(methylsulfonyl)acetamide]), its active metabolite MRE‐269 and various prostacyclin receptor agonists in rat, porcine and human pulmonary arteries

K. Kosugi, K. Tajima, Keiichi Kuwano, 2017, European journal of pharmacology.

A Novel Selective Peroxisome Proliferator-Activated Receptor α Agonist, 2-Methyl-c-5-[4-[5-methyl-2-(4-methylphenyl)-4-oxazolyl]butyl]-1,3-dioxane-r-2-carboxylic acid (NS-220), Potently Decreases Plasma Triglyceride and Glucose Levels and Modifies Lipoprotein Profiles in KK-Ay Mice

J. Yano, M. Murai, T. Asaki, 2004, Journal of Pharmacology and Experimental Therapeutics.

DHCR24 gene knockout mice demonstrate lethal dermopathy with differentiation and maturation defects in the epidermis.

H. Seo, S. Hayasaka, Y. Takagishi, 2006, The Journal of investigative dermatology.

Blockade of voltage-gated calcium channel Cav1.2 and α1-adrenoceptors increases vertebral artery blood flow induced by the antivertigo agent difenidol.

Naoki Inoue, Kohji Murakami, Chiaki Fuchikami, 2012, European journal of pharmacology.