V. Garandel

Mechanisms involved in the inhibition of myoblast proliferation and differentiation by myostatin.

G. Cabello, H. Bernardi, B. Vernus, 2003, Experimental cell research.

Preferential binding of insulin-like growth factor-II (IGF-II) to a putative α2β2 IGF-II receptor type in C2 myoblasts

C. Pinset, D. Montarras, R. Rosenfeld, 1992 .

Insulin-like growth factor I (IGF-I) receptor overexpression abolishes the IGF requirement for differentiation and induces a ligand-dependent transformed phenotype in C2 inducible myoblasts.

J. Schnekenburger, H. Bernardi, B. Barenton, 1997, Endocrinology.

Rabbit slow and fast skeletal muscle‐derived satellite myoblast phenotypes do not involve constitutive differences in the components of the insulin‐like growth factor system

H. Bernardi, F. Bacou, B. Barenton, 1996, Journal of cellular physiology.

A developmentally regulated form of insulin-like growth factor receptor beta-subunit in C2 myoblasts exhibiting altered requirements for differentiation.

R. Garofalo, B. Barenton, V. Garandel, 1993, Endocrinology.

Differentiation of adipocyte precursors in a serum-free medium is influenced by glucocorticoids and endogenously produced insulin-like growth factor-I.

Y. Reyne, J. Nouguès, B. Barenton, 1993, International journal of obesity and related metabolic disorders : journal of the International Association for the Study of Obesity.

Mechanisms involved in the inhibition of myoblast proliferation and differentiation by myostatin.

G. Cabello, H. Bernardi, B. Vernus, 2003, Experimental cell research.