J. O'neil

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Phospholipids in oxidized LDL not adducted to apoB are recognized by the CD36 scavenger receptor.

E. Podrez, G. Hoppe, H. Hoff, 2003, Free radical biology & medicine.

Oxidation of LDL: Role in atherogenesis

H. Hoff, J. O'neil, 1991, Klinische Wochenschrift.

Structural and functional changes in LDL after modification with both 4-hydroxynonenal and malondialdehyde.

H. Hoff, J. O'neil, 1993, Journal of lipid research.

Macrophage degradation of LDL extracted from human aortic plaques: effect of isolation conditions.

H. Hoff, J. O'neil, T. Cole, 1991, Experimental and molecular pathology.

Modification of low density lipoprotein with 4-hydroxynonenal induces uptake by macrophages.

O. Quehenberger, G. Chisolm, H. Esterbauer, 1989, Arteriosclerosis.

Inactivation of cathepsin B by oxidized LDL involves complex formation induced by binding of putative reactive sites exposed at low pH to thiols on the enzyme.

L. Sayre, G. Hoppe, H. Hoff, 1997, Free radical biology & medicine.

Quantification of apo[a] and apoB in human atherosclerotic lesions.

H. Hoff, J. Pepin, J. O'neil, 1991, Journal of lipid research.

Products of lipid peroxidation induce missorting of the principal lysosomal protease in retinal pigment epithelium.

J. Sears, G. Hoppe, H. Hoff, 2004, Biochimica et biophysica acta.

Accumulation of oxidized lipid-protein complexes alters phagosome maturation in retinal pigment epithelium

J. Sears, G. Hoppe, H. Hoff, 2004, Cellular and Molecular Life Sciences CMLS.

Phospholipids in oxidized low density lipoproteins perturb the ability of macrophages to degrade internalized macromolecules and reduce intracellular cathepsin B activity.

G. Hoppe, H. Hoff, J. O'neil, 2003, Atherosclerosis.

Inactivation of lysosomal proteases by oxidized low density lipoprotein is partially responsible for its poor degradation by mouse peritoneal macrophages.

G. Hoppe, H. Hoff, J. O'neil, 1994, The Journal of clinical investigation.

Extracts of human atherosclerotic lesions can modify low density lipoproteins leading to enhanced uptake by macrophages.

H. Hoff, J. O'neil, 1988, Atherosclerosis.

Relationship among antioxidant activity, vasodilation capacity, and phenolic content of red wines.

M. Lean, M. Maclean, A. Crozier, 2000, Journal of agricultural and food chemistry.

Identification of procalcitonin in a rat medullary thyroid carcinoma cell line.

D. M. Burns, B. Roos, W. Mahoney, 1984, The Journal of biological chemistry.

Calcitonin COOH-terminal cleavage peptide as a model for identification of novel neuropeptides predicted by recombinant DNA analysis.

S. Amara, R. Evans, M. Rosenfeld, 1982, The Journal of biological chemistry.

Partial characterization of lipoproteins containing apo[a] in human atherosclerotic lesions.

H. Hoff, A. Yashiro, J. O'neil, 1993, Journal of lipid research.

A non-calcitonin secretory peptide derived from preprocalcitonin.

D. Aron, B. Roos, R. Birnbaum, 1982, The Journal of biological chemistry.

Hydroxynonenal inactivates cathepsin B by forming Michael adducts with active site residues

M. Miyagi, J. Crabb, K. West, 2002, Protein science : a publication of the Protein Society.

Measurement of oxidation in plasma Lp(a) in CAPD patients using a novel ELISA.

E. Podrez, R. G. Salomon, H. Hoff, 1998, Kidney international.

Eubacterial components similar to small nuclear ribonucleoproteins: identification of immunoprecipitable proteins and capped RNAs in a cyanobacterium and a gram-positive eubacterium

J. O'neil, S. A. Kovacs, J. Watcharapijarn, 1993, Journal of bacteriology.

Multiple forms of human tumor calcitonin demonstrated by denaturing polyacrylamide gel electrophoresis and lectin affinity chromatography.

B. Roos, S. Baylin, J. O'neil, 1981, The Journal of clinical endocrinology and metabolism.

Insoluble complex formation of lipoprotein (a) with low density lipoprotein in the presence of calcium ions.

H. Hoff, A. Yashiro, J. O'neil, 1993, The Journal of biological chemistry.

(Carboxyalkyl)pyrroles in human plasma and oxidized low-density lipoproteins.

R. G. Salomon, H. Hoff, K. Kaur, 1997, Chemical research in toxicology.

Phospholipid Hydroxyalkenals: Biological and Chemical Properties of Specific Oxidized Lipids Present in Atherosclerotic Lesions

G. Hoppe, H. Hoff, Zhiping Wu, 2003, Arteriosclerosis, thrombosis, and vascular biology.

Immunochemically detectable lipid-free apo(a) in plasma and in human atherosclerotic lesions.

G. Smejkal, H. Hoff, A. Yashiro, 1994, Chemistry and physics of lipids.

SOME BIOCHEMICAL PROPERTIES OF CHINESE HAMSTER CELLS SENSITIVE AND RESISTANT TO ACTINOMYCIN D

J. L. Biedler, R. Peterson, J. O'neil, 1974, The Journal of cell biology.

Levuglandin E2-protein adducts in human plasma and vasculature.

M. Smith, V. Monnier, R. G. Salomon, 1997, Chemical research in toxicology.

Macrophage uptake of cholesterol-containing particles derived from LDL and isolated from atherosclerotic lesions.

H. Hoff, J. Pepin, J. O'neil, 1990, European heart journal.

Aggregation as well as chemical modification of LDL during oxidation is responsible for poor processing in macrophages.

H. Hoff, D. Armstrong, J. O'neil, 1993, Journal of lipid research.

Protein Adducts of Iso[4]levuglandin E2, a Product of the Isoprostane Pathway, in Oxidized Low Density Lipoprotein*

L. Roberts, K. Kaur, C. Brame, 1999, The Journal of Biological Chemistry.

Lesion-derived low density lipoprotein and oxidized low density lipoprotein share a lability for aggregation, leading to enhanced macrophage degradation.

H F Hoff, J O'Neil, H. Hoff, 1991, Arteriosclerosis and thrombosis : a journal of vascular biology.

Macrophage Receptors Responsible for Distinct Recognition of Low Density Lipoprotein Containing Pyrrole or Pyridinium Adducts: Models of Oxidized Low Density Lipoprotein

E. Podrez, N. Sheibani, L. Sayre, 2022 .

Non-conventional modification of low density lipoproteins: chemical models for macrophage recognition of oxidized LDL.

L. Sayre, J. O'Neil, G. Hoppe, 1997, Biochimica et biophysica acta.

Macrophage recognition of LDL modified by levuglandin E2, an oxidation product of arachidonic acid.

J. O'Neil, G. Hoppe, R. G. Salomon, 1997, Biochimica et biophysica acta.

Lipoprotein(a) Is an Independent Risk Factor for Cardiovascular Disease in Hemodialysis Patients

R. Heyka, H. Hoff, E. Paganini, 1992, Circulation.

Oxidation of low density lipoprotein leads to particle aggregation and altered macrophage recognition.

T. E. Whitaker, H F Hoff, J O'Neil, 1992, The Journal of biological chemistry.

Serum Lp(a) level as a predictor of vein graft stenosis after coronary artery bypass surgery in patients.

G. Beck, B. Lytle, H. Hoff, 1988, Circulation.

Lp(a) and premature mortality during chronic hemodialysis treatment.

H. Hoff, Doris A. Abood, M. Cressman, 1994, Chemistry and physics of lipids.

Oxidation of low-density lipoproteins produces levuglandin-protein adducts.

R. G. Salomon, H. Hoff, G. Subbanagounder, 1997, Chemical research in toxicology.

Plasma immunoreactive calcitonin in lung cancer.

B. Roos, A. Frelinger, S. Baylin, 1980, Endocrine Research Communications.

[End groups of anaphylatoxin. A simple sequence analysis of carboxyl-terminal amino acids].

H. Stegemann, J. O'neil, G. Bernhard, 1964, Hoppe-Seyler's Zeitschrift fur physiologische Chemie.