Zonghui Zhang

发表

EGCG protects against homocysteine-induced human umbilical vein endothelial cells apoptosis by modulating mitochondrial-dependent apoptotic signaling and PI3K/Akt/eNOS signaling pathways

Jinyong Peng, Zeyao Tang, Jingjun Wu, 2017, Apoptosis.

Serum Uric Acid Levels Mediated Atrial Fibrillation in Patients with Hypertension

Maimaiti Yisireyili, Y. Zhao, A. Mahemuti, 2019 .

Phosphocreatine protects endothelial cells from Methylglyoxal induced oxidative stress and apoptosis via the regulation of PI3K/Akt/eNOS and NF-κB pathway.

Jinyong Peng, Zeyao Tang, Shumin Liu, 2017, Vascular pharmacology.

Anticancer effect of SZC015 on lung cancer cells through ROS-dependent apoptosis and autophagy induction mechanisms in vitro.

Jinyong Peng, Zeyao Tang, Lei Gao, 2016, International immunopharmacology.

Erratum to: Phosphocreatine protects against LPS-induced human umbilical vein endothelial cell apoptosis by regulating mitochondrial oxidative phosphorylation

Jinyong Peng, Zeyao Tang, F. Piao, 2016, Apoptosis.

Phosphocreatine protects endothelial cells from oxidized low-density lipoprotein-induced apoptosis by modulating the PI3K/Akt/eNOS pathway

Jinyong Peng, Zeyao Tang, A. Padhiar, 2015, Apoptosis.

Phosphocreatine protects against LPS-induced human umbilical vein endothelial cell apoptosis by regulating mitochondrial oxidative phosphorylation

Jinyong Peng, Zeyao Tang, F. Piao, 2015, Apoptosis.

Erratum to: Phosphocreatine protects against LPS-induced human umbilical vein endothelial cell apoptosis by regulating mitochondrial oxidative phosphorylation

Jinyong Peng, Zeyao Tang, F. Piao, 2016, Apoptosis.

Phosphocreatine protects endothelial cells from oxidized low-density lipoprotein-induced apoptosis by modulating the PI3K/Akt/eNOS pathway

Jinyong Peng, Zeyao Tang, A. Padhiar, 2015, Apoptosis.

Protective effects of phosphocreatine on human vascular endothelial cells against hydrogen peroxide-induced apoptosis and in the hyperlipidemic rat model.

Zeyao Tang, Jiaqi Wang, Jun Wang, 2023, Chemico-biological interactions.