Peng Du

发表

Metabolic wastes are extracellularly disposed by excretosomes, nanotubes and exophers in mouse HT22 cells through an autophagic vesicle clustering mechanism

D. Cui, H. Fu, Weilin Jin, 2019, bioRxiv.

Senile plaques in Alzheimer’s disease arise from Aβ and Cathepsin D-enriched amyloidogenic mixtures out of intravascular hemolysis and Charcot-Bouchard aneurysm-related vascular degeneration

Guo Gao, H. Fu, Weilin Jin, 2022, bioRxiv.

Single-cell Expression Atlas Reveals Cell Heterogeneity in the Creeping Fat of Crohn's Disease.

Xiaolei Wang, Deji Zhuoma, Pengyu Yang, 2023, Inflammatory bowel diseases.

Senile plaques in Alzheimer’s disease arise from Aβ and Cathepsin D-enriched amyloidogenic mixtures out of intravascular hemolysis and Charcot-Bouchard aneurysm-related vascular degeneration

Guo Gao, D. Cui, H. Fu, 2020, bioRxiv.

Senile plaques in Alzheimer's disease arise from Aβ‐ and Cathepsin D‐enriched mixtures leaking out during intravascular haemolysis and microaneurysm rupture

Guo Gao, H. Fu, Weilin Jin, 2022, FEBS letters.

Re-evaluating the nuclear staining on pathological sections in Alzheimer’s disease

Guo Gao, H. Fu, Chunlei Zhang, 2023, bioRxiv.

Senile plaques in Alzheimer’s disease arise from Cathepsin D and Aβ-enriched proteinaceous mixtures out of hemolysis and vascular degeneration

Guo Gao, D. Cui, H. Fu, 2020 .

Aβ-Aggregation-Generated Blue Autofluorescence Illuminates Senile Plaques as well as Complex Blood and Vascular Pathologies in Alzheimer's Disease.

H. Fu, Jilong Li, Peng Du, 2024, Neuroscience bulletin.

Aβ-aggregation-generated blue autofluorescence illuminates senile plaques, complex blood and vascular pathologies in the Alzheimer’s disease

Guo Gao, H. Fu, Chunlei Zhang, 2023, bioRxiv.