J. Dow

Trichloroethylene induced vitamin B(12) and folate deficiency leads to increased formic acid excretion in the rat.

T. Green, J. Dow, Trevor Green, 2000, Toxicology.

The role of glutathione conjugation in the development of kidney tumours in rats exposed to trichloroethylene.

M. Ellis, J. Odum, J. Foster, 1997, Chemico-biological interactions.

Formic acid excretion in rats exposed to trichloroethylene: a possible explanation for renal toxicity in long-term studies.

P. Hext, J. Foster, T. Green, 1998, Toxicology.

Biological monitoring of kidney function among workers occupationally exposed to trichloroethylene

L. Bloemen, C. Ong, T. Green, 2004, Occupational and Environmental Medicine.

An integrated functional genomic study of acute phenobarbital exposure in the rat

Julian L Griffin, J. Griffin, C. Waterfield, 2010, BMC Genomics.

Increased formic acid excretion and the development of kidney toxicity in rats following chronic dosing with trichloroethanol, a major metabolite of trichloroethylene.

J. Foster, T. Green, J. Dow, 2003, Toxicology.