Miranda L. Hanson

Prenatal cadmium exposure alters postnatal immune cell development and function.

J. Barnett, I. Holásková, R. Schafer, 2012, Toxicology and applied pharmacology.

Prenatal cadmium exposure dysregulates sonic hedgehog and Wnt/beta-catenin signaling in the thymus resulting in altered thymocyte development.

J. Barnett, R. Schafer, K. Brundage, 2010, Toxicology and applied pharmacology.

Interleukin-27 Is a Potential Rescue Therapy for Acute Severe Colitis Through Interleukin-10–Dependent, T-Cell–Independent Attenuation of Colonic Mucosal Innate Immune Responses

M. Anver, S. Durum, C. Andrews, 2017, Inflammatory bowel diseases.

Interleukin 27 is a potential rescue therapy for acute severe colitis via interleukin-10 dependent , T 1 cell independent attenuation of colonic mucosal innate immune responses 2

M. Anver, S. Durum, C. Andrews, 2017 .

Oral delivery of IL-27 recombinant bacteria attenuates immune colitis in mice.

M. Anver, S. Durum, Wenqing Li, 2014, Gastroenterology.

Prenatal cadmium exposure produces persistent changes to thymus and spleen cell phenotypic repertoire as well as the acquired immune response.

J. Barnett, I. Holásková, R. Schafer, 2012, Toxicology and applied pharmacology.

Subcellular Localization of the Amide Class Herbicide 3,4-Dichloropropionanilide (DCPA) in T Cells and Hepatocytes

J. Barnett, S. Eremin, C. Peer, 2009, Journal of toxicology and environmental health. Part A.