Elizabeth Keramaris

发表

Adeno-associated virus 9 mediated FKRP gene therapy restores functional glycosylation of α-dystroglycan and improves muscle functions.

Xiao Xiao, Peijuan Lu, Q. Lu, 2013, Molecular therapy : the journal of the American Society of Gene Therapy.

Calpains Mediate p53 Activation and Neuronal Death Evoked by DNA Damage*

David S. Park, R. Slack, M. O'hare, 2003, Journal of Biological Chemistry.

Required Roles of Bax and JNKs in Central and Peripheral Nervous System Death of Retinoblastoma-deficient Mice*

David S. Park, R. Flavell, R. Slack, 2008, Journal of Biological Chemistry.

Ataxia Telangiectasia-mutated Protein Can Regulate p53 and Neuronal Death Independent of Chk2 in Response to DNA Damage*

David S. Park, T. Mak, A. Hirao, 2003, Journal of Biological Chemistry.

Mouse models of fukutin-related protein mutations show a wide range of disease phenotypes

Xiao Xiao, Q. Lu, D. Cowley, 2013, Human Genetics.

Expression of glycosylated α‐dystroglycan in newborn skeletal and cardiac muscles of fukutin related protein (FKRP) mutant mice

J. Tucker, Peijuan Lu, Q. Lu, 2017, Muscle & nerve.

Restoration of Functional Glycosylation of α-Dystroglycan in FKRP Mutant Mice Is Associated with Muscle Regeneration.

Bo Wu, J. Tucker, Q. Lu, 2015, The American journal of pathology.

Adeno-associated virus-mediated overexpression of LARGE rescues α-dystroglycan function in dystrophic mice with mutations in the fukutin-related protein.

Xiao Xiao, Peijuan Lu, Q. Lu, 2014, Human gene therapy methods.

c-Jun N-terminal Kinase 3 Deficiency Protects Neurons from Axotomy-induced Death in Vivo through Mechanisms Independent of c-Jun Phosphorylation*

David S. Park, R. Flavell, R. Slack, 2005, Journal of Biological Chemistry.

Involvement of Caspase 3 in Apoptotic Death of Cortical Neurons Evoked by DNA Damage

David S. Park, G. Robertson, M. Taketo, 2000, Molecular and Cellular Neuroscience.

Interaction of the c-Jun/JNK Pathway and Cyclin-dependent Kinases in Death of Embryonic Cortical Neurons Evoked by DNA Damage*

David S. Park, R. Flavell, R. Slack, 2002, The Journal of Biological Chemistry.

Cyclin-Dependent Kinases and P53 Pathways Are Activated Independently and Mediate Bax Activation in Neurons after DNA Damage

David S. Park, N. Dyson, E. Morris, 2001, The Journal of Neuroscience.

E2F1 Mediates Death of B-amyloid-treated Cortical Neurons in a Manner Independent of p53 and Dependent on Bax and Caspase 3*

David S. Park, G. Robertson, E. Morris, 2000, The Journal of Biological Chemistry.

Involvement of Retinoblastoma Family Members and E2F/DP Complexes in the Death of Neurons Evoked by DNA Damage

David S. Park, H. M. Geller, L. Greene, 2000, The Journal of Neuroscience.

Involvement of Cell Cycle Elements, Cyclin-dependent Kinases, pRb, and E2F·DP, in B-amyloid-induced Neuronal Death*

David S. Park, R. Slack, A. Giovanni, 1999, The Journal of Biological Chemistry.

Mouse models of fukutin-related protein mutations show a wide range of disease phenotypes

Xiao Xiao, Q. Lu, D. Cowley, 2013, Human Genetics.