E. McAdam

The role of secondary bile acids in neoplastic development in the oesophagus.

G. Jenkins, J. Baxter, J. Cronin, 2010, Biochemical Society transactions.

Inducible nitric oxide synthase (iNOS) and nitric oxide (NO) are important mediators of reflux-induced cell signalling in esophageal cells.

J. Baxter, A. Griffiths, Z. Eltahir, 2012, Carcinogenesis.

Epidermal Growth Factor Receptor (EGFR) Is Overexpressed in High‐Grade Dysplasia and Adenocarcinoma of the Esophagus and May Represent a Biomarker of Histological Progression in Barrett's Esophagus (BE)

G. Jenkins, J. Baxter, J. Cronin, 2011 .

Reflux composition influences the level of NF‐κB activation and upstream kinase preference in oesophageal adenocarcinoma cells

G. Jenkins, J. Baxter, A. Griffiths, 2014, International journal of cancer.

Oxidative Stress–Induced Protein Damage Inhibits DNA Repair and Determines Mutation Risk and Therapeutic Efficacy

P. Karran, R. Brem, E. McAdam, 2016, Molecular Cancer Research.

DNA Damage and Repair OxidativeStress – InducedProteinDamage Inhibits DNA Repair and Determines Mutation Risk and Therapeutic Ef fi cacy

P. Karran, R. Brem, E. McAdam, 2016 .

Human papillomavirus infection in Barrett's oesophagus in the UK: an infrequent event.

A. Fiander, G. Jenkins, S. Hibbitts, 2008, Journal of clinical virology : the official publication of the Pan American Society for Clinical Virology.

S1939 Oral Curcumin Suppresses NF-κB Activity in Barrett's Esophagus: A Pilot Study

G. Jenkins, J. Baxter, J. Cronin, 2009 .

Curcumin abrogates bile-induced NF-κB activity and DNA damage in vitro and suppresses NF-κB activity whilst promoting apoptosis in vivo, suggesting chemopreventative potential in Barrett’s oesophagus

G. Jenkins, J. Baxter, J. Cronin, 2012, Clinical and Translational Oncology.