E. Joly

发表

Beta cell compensation for insulin resistance in Zucker fatty rats: increased lipolysis and fatty acid signalling

J. Leahy, M. Prentki, J. Moibi, 2006, Diabetologia.

Selected plant species from the Cree pharmacopoeia of northern Quebec possess anti-diabetic potential.

M. Prentki, S. Bennett, C. Harris, 2006, Canadian journal of physiology and pharmacology.

Beta Cell Failure in Diet-Induced Obese mice stratified according to body weight gain : secretory dysfunction and altered islet lipid metabolism without steatosis or reduced beta cell mass

M. Prentki, S. Madiraju, M. Peyot, 2010 .

β-Cell Failure in Diet-Induced Obese Mice Stratified According to Body Weight Gain: Secretory Dysfunction and Altered Islet Lipid Metabolism Without Steatosis or Reduced β-Cell Mass

M. Prentki, S. Madiraju, M. Peyot, 2010, Diabetes.

Adipose ABHD6 regulates tolerance to cold and thermogenic programs

M. Prentki, I. Chénier, A. Carpentier, 2020, JCI insight.

Pioglitazone Acutely Reduces Energy Metabolism and Insulin Secretion in Rats

M. Prentki, N. Ruderman, V. Poitout, 2013, Diabetes.

Saturated Fatty Acids Synergize with Elevated Glucose to Cause Pancreatic (cid:1) -Cell Death

M. Prentki, G. Dbaibo, J. Buteau, 2003 .

EIF4A2 is a positional candidate gene at the 3q27 locus linked to type 2 diabetes in French families.

T. Hansen, O. Pedersen, C. Dina, 2006, Diabetes.

Adenosine 5'-monophosphate-activated protein kinase and p38 mitogen-activated protein kinase participate in the stimulation of glucose uptake by dinitrophenol in adult cardiomyocytes.

M. Prentki, A. Pelletier, L. Coderre, 2005, Endocrinology.

α/β-Hydrolase Domain 6 Deletion Induces Adipose Browning and Prevents Obesity and Type 2 Diabetes.

M. Prentki, S. Fulton, J. M. Brown, 2016, Cell reports.

A Role for ATP-Citrate Lyase, Malic Enzyme, and Pyruvate/Citrate Cycling in Glucose-induced Insulin Secretion*

M. Prentki, S. Madiraju, C. Guay, 2007, Journal of Biological Chemistry.

A beta cell ATGL-lipolysis/adipose tissue axis controls energy homeostasis and body weight via insulin secretion in mice

M. Prentki, B. Simons, Shupei Wang, 2016, Diabetologia.

Identification of a mammalian glycerol-3-phosphate phosphatase: Role in metabolism and signaling in pancreatic β-cells and hepatocytes

M. Prentki, A. Seifried, J. Iglesias, 2016, Proceedings of the National Academy of Sciences.

Islet beta cell failure in the 60% pancreatectomised obese hyperlipidaemic Zucker fatty rat: severe dysfunction with altered glycerolipid metabolism without steatosis or a falling beta cell mass

J. Leahy, M. Prentki, M. Latour, 2009, Diabetologia.

α/β-Hydrolase domain-6 and saturated long chain monoacylglycerol regulate insulin secretion promoted by both fuel and non-fuel stimuli

M. Prentki, J. Iglesias, S. Madiraju, 2015, Molecular metabolism.

Simplified assays of lipolysis enzymes for drug discovery and specificity assessment of known inhibitors

M. Prentki, K. Skorey, J. Iglesias, 2016, Journal of Lipid Research.

Antidiabetic Activity of Nigella sativa. Seed Extract in Cultured Pancreatic β-cells, Skeletal Muscle Cells, and Adipocytes

M. Prentki, C. Leduc, P. Haddad, 2008 .

Simplified assays of lipolysis enzymes for drug discovery and specificity assessment of of known inhibitors Running title : Lipases and specificity of inhibitors

K. Skorey, J. Iglesias, S. Madiraju, 2015 .

Supplemental Information /-Hydrolase Domain-6-Accessible Monoacylglycerol Controls Glucose-Stimulated Insulin Secretion

M. Prentki, J. M. Brown, J. Iglesias, 2014 .

Malonyl‐CoA decarboxylase is present in the cytosolic, mitochondrial and peroxisomal compartments of rat hepatocytes

M. Prentki, N. Ruderman, A. Saha, 2005, FEBS letters.

Adipose Triglyceride Lipase Is Implicated in Fuel- and Non-fuel-stimulated Insulin Secretion*

M. Prentki, R. Zechner, N. Ruderman, 2009, The Journal of Biological Chemistry.

Malonyl-CoA signaling, lipid partitioning, and glucolipotoxicity: role in beta-cell adaptation and failure in the etiology of diabetes.

M. Prentki, R. Roduit, W. El-Assaad, 2002, Diabetes.

Insulin II Gene Promoter does not Mediate its Transcriptional

M. Ferdaoussi, A. Volchuk, V. Poitout, 2011 .

Oleate Promotes the Proliferation of Breast Cancer Cells via the G Protein-coupled Receptor GPR40*

M. Prentki, É. Joly, Y. Langelier, 2005, Journal of Biological Chemistry.

Saturated Fatty Acid-induced Apoptosis in MDA-MB-231 Breast Cancer Cells

Marc Prentki, M. Prentki, E. Przybytkowski, 2003, Journal of Biological Chemistry.

Upregulation of cellular triacylglycerol - free fatty acid cycling by oleate is associated with long-term serum-free survival of human breast cancer cells.

M. Prentki, E. Przybytkowski, Y. Langelier, 2007, Biochemistry and cell biology = Biochimie et biologie cellulaire.

Glucagon-Like Peptide 1 Induces Pancreatic β-Cell Proliferation Via Transactivation of the Epidermal Growth Factor Receptor

M. Prentki, J. Buteau, S. Foisy, 2003 .

Pancreatic β-Cell Dysfunction in Diet-Induced Obese Mice: Roles of AMP-Kinase, Protein Kinase Cε, Mitochondrial and Cholesterol Metabolism, and Alterations in Gene Expression

M. Prentki, R. Sladek, N. Ruderman, 2016, PloS one.

Metabolic fate of glucose and candidate signaling and excess-fuel detoxification pathways in pancreatic β-cells

M. Prentki, B. Corkey, S. Madiraju, 2017, The Journal of Biological Chemistry.

A Role for Cytosolic Isocitrate Dehydrogenase as a Negative Regulator of Glucose Signaling for Insulin Secretion in Pancreatic ß-Cells

M. Prentki, H. Brunengraber, S. Madiraju, 2013, PloS one.

Saturated fatty acids synergize with elevated glucose to cause pancreatic beta-cell death.

M. Prentki, G. Dbaibo, J. Buteau, 2003, Endocrinology.

β-Arrestin1-mediated recruitment of c-Src underlies the proliferative action of glucagon-like peptide-1 in pancreatic β INS832/13 cells

M. Prentki, J. Buteau, E. Joly, 2012, Molecular and Cellular Endocrinology.

Monoacylglycerol as a metabolic coupling factor in glucose-stimulated insulin secretion

M. Prentki, J. Iglesias, S. Madiraju, 2013 .

An improved PCR-mutagenesis strategy for two-site mutagenesis or sequence swapping between related genes.

E. Joly, É. Joly, R. Kirsch, 1998, Nucleic acids research.

Glucagon-like peptide-1 prevents beta cell glucolipotoxicity

M. Prentki, J. Buteau, L. Rosenberg, 2004, Diabetologia.

Identification of the signals for glucose-induced insulin secretion in INS1 (832/13) β-cells using metformin-induced metabolic deceleration as a model

M. Prentki, B. Corkey, S. Madiraju, 2017, The Journal of Biological Chemistry.

α/β-Hydrolase domain-6-accessible monoacylglycerol controls glucose-stimulated insulin secretion.

M. Prentki, J. M. Brown, J. Iglesias, 2014, Cell metabolism.

Short‐chain 3‐hydroxyacyl‐CoA dehydrogenase is a negative regulator of insulin secretion in response to fuel and non‐fuel stimuli in INS832/13 β‐cells

M. Prentki, S. Madiraju, C. Guay, 2010, Journal of diabetes.

Hormone-sensitive lipase has a role in lipid signaling for insulin secretion but is nonessential for the incretin action of glucagon-like peptide 1.

M. Prentki, G. Mitchell, B. Corkey, 2004, Diabetes.

Glucolipotoxicity alters lipid partitioning and causes mitochondrial dysfunction, cholesterol, and ceramide deposition and reactive oxygen species production in INS832/13 ss-cells.

M. Prentki, R. Sladek, J. Buteau, 2010, Endocrinology.

Pioglitazone acutely reduces insulin secretion and causes metabolic deceleration of the pancreatic beta-cell at submaximal glucose concentrations.

M. Prentki, N. Ruderman, V. Poitout, 2009, Endocrinology.

Defective insulin secretory response to intravenous glucose in C57Bl/6J compared to C57Bl/6N mice

M. Prentki, T. Alquier, V. Poitout, 2014, Molecular metabolism.

Glucose represses PPARαgene expression via AMP‐activated protein kinase but not via p38 mitogen‐activated protein kinase in the pancreatic β‐cell

M. Prentki, N. Ruderman, L. Witters, 2009, Journal of diabetes.

Role of transcription factor KLF11 and its diabetes-associated gene variants in pancreatic beta cell function.

T. Hansen, O. Pedersen, K. Clément, 2005, Proceedings of the National Academy of Sciences of the United States of America.

Differential Insulin Secretion of High-Fat Diet-Fed C57BL/6NN and C57BL/6NJ Mice: Implications of Mixed Genetic Background in Metabolic Studies

M. Prentki, S. Madiraju, Dongwei Zhang, 2016, PloS one.

Glucagon-like peptide 1 induces pancreatic beta-cell proliferation via transactivation of the epidermal growth factor receptor.

M. Prentki, J. Buteau, S. Foisy, 2003, Diabetes.

THE BRET2/ARRESTIN ASSAY IN STABLE RECOMBINANT CELLS: A PLATFORM TO SCREEN FOR COMPOUNDS THAT INTERACT WITH G PROTEIN-COUPLED RECEPTORS (GPCRS)*

S. Angers, B. Houle, L. Bertrand, 2002, Journal of receptor and signal transduction research.

Bret2: Efficient Energy Transfer from Renilla Luciferase to GFP2 to Measure Protein-Protein Interactions and Intracellular Signaling Events in Live Cells

Sean Taylor, B. Houle, A. Labonté, 2001 .

Binding of activating transcription factor 6 to the A5/Core of the rat insulin II gene promoter does not mediate its transcriptional repression.

M. Ferdaoussi, A. Volchuk, V. Poitout, 2011, Journal of molecular endocrinology.

Adenosine 5 (cid:1) -Monophosphate-Activated Protein Kinase and p38 Mitogen-Activated Protein Kinase Participate in the Stimulation of Glucose Uptake by Dinitrophenol in Adult Cardiomyocytes

M. Prentki, A. Pelletier, L. Coderre, 2005 .