A. Planagumà

发表

Liver: the formation and actions of aspirin-triggered lipoxins.

J. Clària, A. Planagumà, 2005, Prostaglandins, leukotrienes, and essential fatty acids.

Inhibition of 5‐lipoxygenase‐activating protein abrogates experimental liver injury: role of Kupffer cells

V. Arroyo, J. Clària, R. Miquel, 2005, Journal of leukocyte biology.

Aspirin (ASA) regulates 5‐lipoxygenase activity and peroxisome proliferator‐activated receptor α‐mediated CINC‐1 release in rat liver cells: novel actions of lipoxin A4 (LXA4) and ASA‐triggered 15‐epi‐LXA4

V. Arroyo, J. Clària, E. Titos, 2002, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.

Lovastatin Decreases Acute Mucosal Inflammation Via 15-epi-Lipoxin A4

B. Levy, C. Serhan, M. Pfeffer, 2010, Mucosal Immunology.

Lack of activity of 15‐epi‐lipoxin A4 on FPR2/ALX and CysLT1 receptors in interleukin‐8‐driven human neutrophil function

S. Sentellas, R. Malhotra, M. Miralpeix, 2013, Clinical and experimental immunology.

5-Lipoxygenase (5-LO) is Involved in Kupffer Cell Survival. Possible Role of 5-LO Products in the Pathogenesis of Liver Fibrosis

N. Villamor, V. Arroyo, F. Rivera, 2004, Comparative Hepatology.

5-Lipoxygenase (5-LO) is Involved in Kupffer Cell Survival. Possible Role of 5-LO Products in the Pathogenesis of Liver Fibrosis.

N. Villamor, W. Jiménez, V. Arroyo, 2002, Comparative hepatology.

Docosahexaenoic acid (DHA) blunts liver injury by conversion to protective lipid mediators: protectin D1 and 17Sâ•’hydroxyâ•’DHA

V. Arroyo, J. Rodés, K. Gronert, 2006 .

Combined anti CXC receptors 1 and 2 therapy is a promising anti-inflammatory treatment for respiratory diseases by reducing neutrophil migration and activation.

N. Prats, M. Lehner, V. Segarra, 2015, Pulmonary pharmacology & therapeutics.

Endogenous cannabinoids: a new system involved in the homeostasis of arterial pressure in experimental cirrhosis in the rat.

R. Martı́n-Ruiz, P. Cejudo-Martı́n, V. Arroyo, 2002, Gastroenterology.

The selective cyclooxygenase‐2 inhibitor SC‐236 reduces liver fibrosis by mechanisms involving non‐parenchymal cell apoptosis and PPARγ activation

J. Masferrer, V. Arroyo, J. Clària, 2005, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.

Cyclooxygenase‐1 derived prostaglandins are involved in the maintenance of renal function in rats with cirrhosis and ascites

J. Masferrer, W. Jiménez, V. Arroyo, 2002, British journal of pharmacology.

Renal effects of selective cyclooxygenase inhibition in experimental liver disease.

J. Masferrer, W. Jiménez, V. Arroyo, 2003, Advances in experimental medicine and biology.

Airway lipoxin A4 generation and lipoxin A4 receptor expression are decreased in severe asthma.

W. Busse, E. Bleecker, E. Israel, 2008, American journal of respiratory and critical care medicine.

Comparative Protection against Liver Inflammation and Fibrosis by a Selective Cyclooxygenase-2 Inhibitor and a Nonredox-Type 5-Lipoxygenase Inhibitor

J. Masferrer, V. Arroyo, J. Clària, 2007, Journal of Pharmacology and Experimental Therapeutics.

The selective cyclooxygenase-2 inhibitor SC-236 reduces liver fibrosis by mechanisms involving non-parenchymal cell apoptosis and PPARgamma activation.

J. Masferrer, V. Arroyo, J. Clària, 2005, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.

Docosahexaenoic acid (DHA) blunts liver injury by conversion to protective lipid mediators: protectin D1 and 17S‐hydroxy‐DHA

V. Arroyo, J. Rodés, K. Gronert, 2006, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.

The selective cyclooxygenase-2 inhibitor celecoxib modulates the formation of vasoconstrictor eicosanoids and activates PPARgamma. Influence of albumin.

J. Masferrer, W. Jiménez, V. Arroyo, 2005, Journal of hepatology.

Inhibition of 5‐lipoxygenase induces cell growth arrest and apoptosis in rat Kupffer cells: implications for liver fibrosis

N. Villamor, A. Carrió, W. Jiménez, 2003, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.

Increased susceptibility to exacerbated liver injury in hypercholesterolemic ApoE-deficient mice: potential involvement of oxysterols.

R. Bataller, J. Joven, V. Arroyo, 2009, American journal of physiology. Gastrointestinal and liver physiology.

Comparative Protection Against Liver Inflammation and Fibrosis by a Selective COX-2 Inhibitor and a Nonredox-type 5-LO Inhibitor

J. Masferrer, V. Arroyo, J. Clària, 2007 .

Estradiol and mTORC2 cooperate to enhance prostaglandin biosynthesis and tumorigenesis in TSC2-deficient LAM cells

Jing Li, J. Blenis, E. Israel, 2014, The Journal of experimental medicine.

Exhaled breath condensate eicosanoid levels associate with asthma and its severity.

E. Israel, M. Bonini, B. Levy, 2013, The Journal of allergy and clinical immunology.

Application of a phenotypic drug discovery strategy to identify biological and chemical starting points for inhibition of TSLP production in lung epithelial cells

Rosa López, Estrella Lozoya, Stephane Paris, 2018, PloS one.

Uncontrolled airway inflammation in lung disease represents a defect in counter-regulatory signaling.

B. Levy, A. Planagumà, 2008, Future lipidology.

Gene expression profiling of renal dysfunction in rats with experimental cirrhosis.

V. Arroyo, J. Rodés, J. Clària, 2006, Journal of hepatology.

The selective cyclooxygenase-2 inhibitor celecoxib modulates the formation of vasoconstrictor eicosanoids and activates PPARgamma. Influence of albumin.

J. Masferrer, W. Jiménez, V. Arroyo, 2005, Journal of hepatology.

Airway lipoxin A4 generation and lipoxin A4 receptor expression are decreased in severe asthma.

W. Busse, E. Bleecker, E. Israel, 2008, American journal of respiratory and critical care medicine.

21 The selective cyclooxygenase-2 inhibitor SC-236 reduces experimental liver fibrosis by inactivating kupffer cells and acting as a peroxisome proliferator-activated receptor gamma ligand

J. Masferrer, V. Arroyo, J. Rodés, 2004 .