Xuxiao He

发表

Fructose‐1,6‐Bisphosphate Aldolase B Depletion Promotes Hepatocellular Carcinogenesis Through Activating Insulin Receptor Signaling and Lipogenesis

Yongqiang Wang, H. Yin, Yongzhen Tao, 2021, Hepatology.

Aldolase B suppresses hepatocellular carcinogenesis by inhibiting G6PD and pentose phosphate pathways

Jianping Ding, Yu Li, H. Yin, 2020, Nature Cancer.

Aerobic glycolysis promotes tumor immune evasion by hexokinase2-mediated phosphorylation of IκBα.

Y. Tong, Hongfei Jiang, Jing Fang, 2022, Cell metabolism.

Yeast β-D-glucan exerts antitumour activity in liver cancer through impairing autophagy and lysosomal function, promoting reactive oxygen species production and apoptosis

H. Yin, Hong-zhi Liu, Qiang Wang, 2020, Redox biology.

Loss of STAT5A promotes glucose metabolism and tumor growth through miRNA‐23a‐AKT signaling in hepatocellular carcinoma

H. Yin, Shuqun Cheng, Wei-xing Guo, 2020, Molecular oncology.

Loss of hepatic aldolase B activates Akt and promotes hepatocellular carcinogenesis by destabilizing the Aldob/Akt/PP2A protein complex

G. Narla, H. Yin, Shuqun Cheng, 2020, PLoS biology.

Loss of hepatic aldolase B activates Akt and promotes hepatocellular carcinogenesis by destabilizing Aldob/Akt/PP2A protein complex

G. Narla, H. Yin, Shuqun Cheng, 2020, bioRxiv.

Polyphenolic Proanthocyanidin-B2 suppresses proliferation of liver cancer cells and hepatocellular carcinogenesis through directly binding and inhibiting AKT activity

H. Yin, Qiang Wang, Aimin Shi, 2020, Redox biology.

Fructose-1,6-bisphosphatase 1 dephosphorylates and inhibits TERT for tumor suppression.

Huang Yang, Shudi Luo, Xuxiao He, 2024, Nature chemical biology.