V. Conlin

Fetal mouse NK cell clones are deficient in Ly49 expression, share a common broad lytic specificity, and undergo continuous and extensive diversification in vitro.

M. Manoussaka, C. Brooks, J. Toomey, 1998, Journal of immunology.

SHIP-deficient mice develop spontaneous intestinal inflammation and arginase-dependent fibrosis.

J. Boucher, K. Jacobson, L. Sly, 2011, The American journal of pathology.

Vasoactive intestinal peptide prevents PKCε-induced intestinal epithelial barrier disruption during EPEC infection.

U. Dalwadi, V. Morampudi, K. Jacobson, 2015, American journal of physiology. Gastrointestinal and liver physiology.

Diet, Microbiome, and the Intestinal Epithelium: An Essential Triumvirate?

J. Guzman, C. Jobin, V. Conlin, 2013, BioMed research international.

Vasoactive Intestinal Polypeptide Promotes Intestinal Barrier Homeostasis and Protection Against Colitis in Mice

Y. Nasser, J. Waschek, H. Yu, 2015, PloS one.

Vasoactive intestinal peptide ameliorates intestinal barrier disruption associated with Citrobacter rodentium-induced colitis.

K. Jacobson, B. Vallance, A. Buchan, 2009, American journal of physiology. Gastrointestinal and liver physiology.

Helicobacter pylori Infection Targets Adherens Junction Regulatory Proteins and Results in Increased Rates of Migration in Human Gastric Epithelial Cells

B. Finlay, A. Buchan, E. Moore, 2004, Infection and Immunity.

Tricellular Tight Junction Protein Tricellulin Is Targeted by the Enteropathogenic Escherichia coli Effector EspG1, Leading to Epithelial Barrier Disruption

M. Stahl, U. Dalwadi, H. Yu, 2016, Infection and Immunity.