S. Stahl

发表

Loss of diphthamide pre-activates NF-κB and death receptor pathways and renders MCF7 cells hypersensitive to tumor necrosis factor

I. Pastan, U. Brinkmann, T. Racek, 2015, Proceedings of the National Academy of Sciences.

Cytomegalovirus Downregulates IRE1 to Repress the Unfolded Protein Response

R. Zahedi, A. Sickmann, B. Tirosh, 2013, PLoS pathogens.

Phytoestrogens act as estrogen agonists in an estrogen-responsive pituitary cell line.

T. Chun, W. Gray, S. Stahl, 1998, Toxicology and applied pharmacology.

No functional and transductional significance of specific neuropilin 1 siRNA inhibition in colon carcinoma cell lines lacking VEGF receptor 2.

T. Sauerbruch, V. Schmitz, M. Quante, 2009, Oncology reports.

Factors that Determine Sensitivity and Resistances of Tumor Cells Towards Antibody-Targeted Protein Toxins

I. Pastan, U. Brinkmann, S. Stahl, 2015 .

Influence of DPH1 and DPH5 Protein Variants on the Synthesis of Diphthamide, the Target of ADP-Ribosylating Toxins

U. Brinkmann, Klaus Mayer, Jérôme Schnitger, 2017, Toxins.

Tachykinin NK(2) receptor mediates contraction and ion transport in rat colon by different mechanisms.

C. Maggi, R. Patacchini, H. Cox, 2001, European journal of pharmacology.

Diphthamide affects selenoprotein expression: Diphthamide deficiency reduces selenocysteine incorporation, decreases selenite sensitivity and pre-disposes to oxidative stress

I. Pastan, U. Brinkmann, H. Kettenberger, 2018, Redox biology.

Interplay between reversible phosphorylation and irreversible ADP-ribosylation of eukaryotic translation elongation factor 2

I. Pastan, U. Brinkmann, S. Dengl, 2018, Biological chemistry.