J. Robinson

发表

T Cell–Expressed CD40L Potentiates the Bone Anabolic Activity of Intermittent PTH Treatment

R. Pacifici, M. Weitzmann, Mingcan Yu, 2015, Journal of bone and mineral research : the official journal of the American Society for Bone and Mineral Research.

Dephosphorylation and inactivation of NPR2 guanylyl cyclase in granulosa cells contributes to the LH-induced decrease in cGMP that causes resumption of meiosis in rat oocytes

L. Jaffe, T. Uliasz, Leia C. Shuhaibar, 2014, Development.

Guanylyl cyclase‐A phosphorylation decreases cardiac hypertrophy and improves systolic function in male, but not female, mice

T. O’Connell, J. Osborn, Chastity L. Healy, 2021, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.

Guanylyl Cyclases A and B Are Asymmetric Dimers That Are Allosterically Activated by ATP Binding to the Catalytic Domain

J. Robinson, L. Potter, 2012, Science Signaling.

Activated by ATP Binding to the Catalytic Domain Guanylyl Cyclases A and B Are Asymmetric Dimers That Are Allosterically

J. Robinson, L. Potter, 2012 .

Prevention of guanylyl cyclase–B dephosphorylation rescues achondroplastic dwarfism

Yi-Ching Lee, N. Kaci, L. Legeai-Mallet, 2021, JCI insight.

Male mice with elevated C-type natriuretic peptide-dependent guanylyl cyclase-B activity have increased osteoblasts, bone mass and bone strength.

S. Khosla, L. Suva, D. Gaddy, 2020, Bone.

A human skeletal overgrowth mutation increases maximal velocity and blocks desensitization of guanylyl cyclase-B.

K. Ozono, T. Michigami, D. Dickey, 2013, Bone.

Dephosphorylation is the Mechanism of Fibroblast Growth Factor Inhibition of Guanylyl Cyclase-B

L. Jaffe, H. Schmidt, J. Davydova, 2017, bioRxiv.

Dephosphorylation is the Mechanism of Fibroblast Growth Factor Inhibition of Guanylyl Cyclase-B

L. Jaffe, H. Schmidt, J. Davydova, 2017, bioRxiv.

Dephosphorylation of the NPR2 guanylyl cyclase contributes to inhibition of bone growth by fibroblast growth factor

R. Feil, L. Jaffe, T. Uliasz, 2017, bioRxiv.

The indolocarbazole, Gö6976, inhibits guanylyl cyclase‐A and ‐B

X. Lou, J. Robinson, L. Potter, 2011, British journal of pharmacology.

The indolocarbazole, G6976, inhibits guanylyl cyclaseA and B

X. Lou, J. Robinson, L. Potter, 2011 .

Luteinizing hormone reduces the activity of the NPR2 guanylyl cyclase in mouse ovarian follicles, contributing to the cyclic GMP decrease that promotes resumption of meiosis in oocytes.

L. Jaffe, J. Eppig, Meijia Zhang, 2012, Developmental biology.

Regulation of the Natriuretic Peptide Receptor 2 (Npr2) by Phosphorylation of Juxtamembrane Serine and Threonine Residues Is Essential for Bifurcation of Sensory Axons

R. Feil, Ralf Kühn, H. Schmidt, 2018, The Journal of Neuroscience.

Guanylyl Cyclase-B Dependent Bone Formation in Mice is Associated with Youth, Increased Osteoblasts, and Decreased Osteoclasts

S. Khosla, L. Suva, D. Gaddy, 2022, Calcified Tissue International.

Catalytically Active Guanylyl Cyclase B Requires Endoplasmic Reticulum-mediated Glycosylation, and Mutations That Inhibit This Process Cause Dwarfism*

N. Otto, D. Dickey, J. Robinson, 2016, The Journal of Biological Chemistry.

Heterozygous Mutations in Natriuretic Peptide Receptor‐B (NPR2) Gene as a Cause of Short Stature

Timothy C. Miller, J. Hirschhorn, M. Warman, 2015, Human mutation.

A Functional Screen Provides Evidence for a Conserved, Regulatory, Juxtamembrane Phosphorylation Site in Guanylyl Cyclase A and B

T. Griffin, M. Stone, D. Dickey, 2012, PloS one.

Novel enhancers of guanylyl cyclase-A activity via allosteric modulation

F. Levy, A. Cataliotti, A. Hobbs, 2022, bioRxiv.

ATP Potentiates Competitive Inhibition of Guanylyl Cyclase A and B by the Staurosporine Analog, Gö6976

J. Robinson, L. Potter, 2011, The Journal of Biological Chemistry.

Using Representational Difference Analysis to detect changes in transcript expression of Aiptasia genes after laboratory exposure to lindane.

M. B. Morgan, J. Robinson, C. Parker, 2012, Aquatic toxicology.

Tumor-derived NKG2D ligand sMIC reprograms NK cells to an inflammatory phenotype through CBM signalosome activation

D. Wainwright, Zhe Ji, S. Mackay, 2021, Communications biology.

Abstracts from the 8th International Conference on cGMP Generators, Effectors and Therapeutic Implications

Jordi Villà-Freixa, Peter Sandner, Bernd J. Pichler, 2017, BMC Pharmacology and Toxicology.

Hydrogen Sulfide Is a Novel Regulator of Bone Formation Implicated in the Bone Loss Induced by Estrogen Deficiency

G. Lisignoli, J. Calvert, R. Pacifici, 2016, Journal of bone and mineral research : the official journal of the American Society for Bone and Mineral Research.

Cyclic AMP links luteinizing hormone signaling to dephosphorylation and inactivation of the NPR2 guanylyl cyclase in ovarian follicles†

L. Jaffe, T. Uliasz, J. Egbert, 2021, Biology of Reproduction.

IL-17A Is Increased in Humans with Primary Hyperparathyroidism and Mediates PTH-Induced Bone Loss in Mice.

R. Pacifici, G. Isaia, M. Weitzmann, 2015, Cell metabolism.

Abstracts from the 8th International Conference on cGMP Generators, Effectors and Therapeutic Implications

James C. Campbell, Dong I. Lee, M. Zaccolo, 2017, BMC Pharmacology and Toxicology.

Guanylyl Cyclases A and B Are Asymmetric Dimers That Are Allosterically Activated by ATP Binding to the Catalytic Domain

J. Robinson, L. Potter, 2012, Science Signaling.

IL-17A Is Increased in Humans with Primary Hyperparathyroidism and Mediates PTH-Induced Bone Loss in Mice.

R. Pacifici, G. Isaia, M. Weitzmann, 2015, Cell metabolism.