Jan G. Christian

Impact of Leishmania Infection on Host Macrophage Nuclear Physiology and Nucleopore Complex Integrity

W. McMaster, M. Olivier, U. Stochaj, 2015, PLoS pathogens.

Chlamydia trachomatis can protect host cells against apoptosis in the absence of cellular Inhibitor of Apoptosis Proteins and Mcl-1.

G. Häcker, S. Ying, S. Paschen, 2008, Microbes and infection.

Cleavage of the NF-κB Family Protein p65/RelA by the Chlamydial Protease-like Activity Factor (CPAF) Impairs Proinflammatory Signaling in Cells Infected with Chlamydiae*

G. Häcker, J. Vier, Stefan A. Paschen, 2010, The Journal of Biological Chemistry.

Targeting of a Chlamydial Protease Impedes Intracellular Bacterial Growth

T. Meyer, J. Rupp, G. Häcker, 2011, PLoS pathogens.

Cytopathicity of Chlamydia is largely reproduced by expression of a single chlamydial protease

A. Walch, D. Ojcius, G. Häcker, 2008, The Journal of cell biology.

MYC and EGR1 synergize to trigger tumor cell death by controlling NOXA and BIM transcription upon treatment with the proteasome inhibitor bortezomib

D. Saur, R. Schmid, G. Häcker, 2014, Nucleic acids research.

Raman microspectroscopy reveals long‐term extracellular activity of chlamydiae

M. Wagner, K. Mechtler, M. Horn, 2010, Molecular microbiology.

PKC/ROS-Mediated NLRP3 Inflammasome Activation Is Attenuated by Leishmania Zinc-Metalloprotease during Infection

K. Chang, M. Olivier, M. Shio, 2015, PLoS neglected tropical diseases.