Konstantin Wemhöner

The acid-sensitive potassium channel TASK-1 in rat cardiac muscle.

F. Sachse, E. Wischmeyer, S. Rinné, 2007, Cardiovascular research.

The inhibition of the potassium channel TASK-1 in rat cardiac muscle by endothelin-1 is mediated by phospholipase C.

Vijay Renigunta, N. Decher, G. Schlichthörl, 2013, Cardiovascular research.

Knock-Out of the Potassium Channel TASK-1 Leads to a Prolonged QT Interval and a Disturbed QRS Complex

F. Sachse, S. Meuth, M. Zuzarte, 2011, Cellular Physiology and Biochemistry.

Gain-of-function mutations in the calcium channel CACNA1C (Cav1.2) cause non-syndromic long-QT but not Timothy syndrome.

E. Schulze-Bahr, F. Sachse, A. Coffey, 2015, Journal of molecular and cellular cardiology.

The VAMP-associated protein VAPB is required for cardiac and neuronal pacemaker channel function

C. Culmsee, T. Budde, L. Monassier, 2018, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.

A Leucine Zipper Motif Essential for Gating of Hyperpolarization-activated Channels*

P. Stansfeld, S. Rinné, N. Decher, 2012, The Journal of Biological Chemistry.

An N-terminal deletion variant of HCN1 in the epileptic WAG/Rij strain modulates HCN current densities

S. Meuth, T. Budde, S. Rinné, 2015, Front. Mol. Neurosci..

Knock-Out of the Potassium Channel TASK-1 Leads to a Prolonged QT Interval and a Disturbed QRS Complex

F. Sachse, S. Meuth, S. Rinné, 2011, Cellular Physiology and Biochemistry.

Gain-of-function mutations in the calcium channel CACNA1C (Cav1.2) cause non-syndromic long-QT but not Timothy syndrome.

E. Schulze-Bahr, F. Sachse, A. Coffey, 2015, Journal of molecular and cellular cardiology.