S. Thiessen

Endoplasmic reticulum stress caused by overexpression of islet-specific glucose-6-phosphatase catalytic subunit-related protein in pancreatic Beta-cells.

P. Santamaria, A. Shameli, J. Yamanouchi, 2007, The review of diabetic studies : RDS.

T-cell tolerance by dendritic cells and macrophages as a mechanism for the major histocompatibility complex-linked resistance to autoimmune diabetes.

P. Santamaria, J. Verdaguer, A. Amrani, 2002, Diabetes.

IL-1α, IL-1β, and IFN-γ mark β cells for Fas-dependent destruction by diabetogenic CD4+ T lymphocytes

P. Santamaria, J. Verdaguer, A. Amrani, 2000 .

CD154-dependent priming of diabetogenic CD4(+) T cells dissociated from activation of antigen-presenting cells.

Bingye Han, P. Santamaria, J. Verdaguer, 2002, Immunity.

CD40 ligation releases immature dendritic cells from the control of regulatory CD4+CD25+ T cells.

T. Utsugi, Bingye Han, P. Santamaria, 2003, Immunity.

IL-1alpha, IL-1beta, and IFN-gamma mark beta cells for Fas-dependent destruction by diabetogenic CD4(+) T lymphocytes.

P. Santamaria, J. Verdaguer, A. Amrani, 2000, The Journal of clinical investigation.

Antidiabetogenic MHC class II promotes the differentiation of MHC-promiscuous autoreactive T cells into FOXP3+ regulatory T cells

J. Elliott, P. Santamaria, X. Clemente-Casares, 2013, Proceedings of the National Academy of Sciences.

RAG-dependent peripheral T cell receptor diversification in CD8+ T lymphocytes

Bingye Han, P. Santamaria, A. Amrani, 2002, Proceedings of the National Academy of Sciences of the United States of America.

A T follicular helper cell origin for T regulatory type 1 cells

J. Saez-Rodriguez, C. Fandos, Yang Yang, 2023, Cellular & Molecular Immunology.

IL-1α, IL-1β, and IFN-γ mark β cells for Fas-dependent destruction by diabetogenic CD4+ T lymphocytes

P. Santamaria, J. Verdaguer, A. Amrani, 2000 .