M. Madlener

发表

Transforming growth factors beta1, beta2, and beta3 and their receptors are differentially regulated during normal and impaired wound healing.

S. Werner, S. Frank, M. Madlener, 1996, The Journal of biological chemistry.

Differential expression of matrix metalloproteinases and their physiological inhibitors in acute murine skin wounds

M. Madlener, 1998, Archives of Dermatological Research.

Regulation of the expression of stromelysin-2 by growth factors in keratinocytes: implications for normal and impaired wound healing.

S. Werner, M. Brauchle, W. Parks, 1996, The Biochemical journal.

Keratinocyte growth factor is highly overexpressed in inflammatory bowel disease.

P. Hofschneider, S. Werner, M. Brauchle, 1996, The American journal of pathology.

Matrix metalloproteinases (MMPs) and their physiological inhibitors (TIMPs) are differentially expressed during excisional skin wound repair.

S. Werner, W. Parks, S. Werner, 1998, Experimental cell research.

Induction of inducible nitric oxide synthase and its corresponding tetrahydrobiopterin-cofactor-synthesizing enzyme GTP-cyclohydrolase I during cutaneous wound repair.

S. Werner, J. Pfeilschifter, S Frank, 1998, The Journal of investigative dermatology.

Transforming Growth Factors 1, 2, and 3 and Their Receptors Are Differentially Regulated during Normal and Impaired Wound Healing (*)

S. Werner, S. Frank, M. Madlener, 1996, The Journal of Biological Chemistry.

Differential regulation of pro-inflammatory cytokines during wound healing in normal and glucocorticoid-treated mice.

S. Werner, M. Brauchle, R. Fässler, 1996, Cytokine.

cDNA cloning and expression of the gene encoding murine stromelysin-2 (MMP-10).

S. Werner, M. Madlener, 1997, Gene.