D. Seyidova

Mitochondria DNA deletions in atherosclerotic hypoperfused brain microvessels as a primary target for the development of Alzheimer's disease

G. Perry, Mark A. Smith, Shu G. Chen, 2005, Journal of the Neurological Sciences.

Exploring ischemia-induced vascular lesions and potential pharmacological intervention strategies.

M. Obrenovich, J. C. de la Torre, G. Aliev, 2005, Histology and histopathology.

X-ray contrast media induce aortic endothelial damage, which can be prevented with prior heparin treatment.

J. LaManna, M. Smith, A. Raina, 2003, Journal of submicroscopic cytology and pathology.

The role of nitric oxide in the pathogenesis of brain lesions during the development of Alzheimer's disease.

M. Obrenovich, Mark A. Smith, B. Lamb, 2004, In vivo.

The Role of Oxidative Stress in the Pathophysiology of Cerebrovascular Lesions in Alzheimer's Disease

H. Vinters, J. LaManna, A. Nunomura, 2002, Brain pathology.

Mitochondria and vascular lesions as a central target for the development of Alzheimer's disease and Alzheimer disease-like pathology in transgenic mice

H. Vinters, J. LaManna, M. Obrenovich, 2003, Neurological research.

Atherosclerotic Lesions and Mitochondria DNA Deletions in Brain Microvessels as a Central Target for the Development of Human AD and AD‐Like Pathology in Aged Transgenic Mice

C. Cotman, H. Vinters, J. LaManna, 2002, Annals of the New York Academy of Sciences.

Is nitric oxide a key target in the pathogenesis of brain lesions during the development of Alzheimer's disease?

M. Obrenovich, Mark A. Smith, Shu G. Chen, 2004, Neurological research.

Increased expression of NOS and ET-1 immunoreactivity in human colorectal metastatic liver tumours is associated with selective depression of constitutive NOS immunoreactivity in vessel endothelium.

G. Burnstock, J. LaManna, Mark A. Smith, 2002, Journal of submicroscopic cytology and pathology.