A. M. Ferramola de Sancovich

Hexachlorobenzene, a Dioxin-Type Compound, Increases Malic Enzyme Gene Transcription through a Mechanism Involving the Thyroid Hormone Response Element1.

P. Santisteban, A. Randi, D. L. Kleiman de Pisarev, 1999, Endocrinology.

Hexachlorobenzene, a dioxin-type compound, increases malic enzyme gene transcription through a mechanism involving the thyroid hormone response element.

P. Santisteban, A. Loaiza-Pérez, A. Randi, 1999, Endocrinology.

Studies on the decrease of liver cytochrome P-450 content by triiodothyronine.

D. L. Kleiman de Pisarev, H. A. Sancovich, A. M. Ferramola de Sancovich, 1991, Biochimica et biophysica acta.

Effect of in vivo administered hexachlorobenzene on epidermal growth factor receptor levels, protein tyrosine kinase activity, and phosphotyrosine content in rat liver.

A. Randi, D. L. Kleiman de Pisarev, Rodolfo A. Kölliker Frers, 2003, Biochemical pharmacology.

Hexachlorobenzene-induced alterations of rat hepatic microsomal membrane function.

A. Randi, D. L. Kleiman de Pisarev, L. Krawiec, 1998, Toxicology.

Disturbances on delta aminolevulinate dehydratase (ALA-D) enzyme activity by Pb2+, Cd2+, Cu2+, Mg2+, Zn2+, Na+, K+ and Li+: analysis based on coordination geometry and acid-base Lewis capacity.

H. A. Sancovich, A. M. Ferramola de Sancovich, N. L. Pauza, 2005, Journal of inorganic biochemistry.

Hexachlorobenzene Treatment on Hepatic Mitochondrial Function Parameters and Intracellular Coproporphyrinogen Oxidase Location

H. A. Sancovich, A. M. Ferramola de Sancovich, Y. Sopena, 2008, International journal of toxicology.

The isolation and identification of indigoid pigments from urine.

H. A. Sancovich, A. M. Ferramola de Sancovich, M. Grinstein, 1988, Clinica chimica acta; international journal of clinical chemistry.

Effect of thyroidectomy and thyroxine on hexachlorobenzene induced porphyria

H. A. Sancovich, A. M. Ferramola de Sancovich, D. L. K. Pisarev, 1994, Journal of endocrinological investigation.