Anke Di

发表

Calcium-G Protein Interactions in the Regulation of Macrophage Secretion*

D. Nelson, Anke Di, Boris Krupa, 2001, The Journal of Biological Chemistry.

ROS-activated calcium signaling mechanisms regulating endothelial barrier function.

A. Malik, D. Mehta, Anke Di, 2016, Cell calcium.

Role of the phagosomal redox-sensitive TRP channel TRPM2 in regulating bactericidal activity of macrophages

Haixia Gong, A. Malik, T. Kiya, 2017, Journal of Cell Science.

A unique human blood-derived cell population displays high potential for producing insulin.

Z. Huang, Yong Wang, Yong Zhao, 2007, Biochemical and biophysical research communications.

The TWIK2 Potassium Efflux Channel in Macrophages Mediates NLRP3 Inflammasome‐Induced Inflammation

A. Malik, J. Rehman, T. Kanneganti, 2018, Immunity.

A Novel Function of Sphingosine Kinase 1 Suppression of JNK Activity in Preventing Inflammation and Injury*

Xiao-pei Gao, A. Malik, Haiyang Tang, 2010, The Journal of Biological Chemistry.

A Process for Controlling Intracellular Bacterial Infections Induced by Membrane Injury

J. Bliska, N. Andrews, D. Nelson, 2004, Science.

Caspase-11–mediated endothelial pyroptosis underlies endotoxemia-induced lung injury

A. Malik, J. Rehman, Z. Hong, 2017, The Journal of clinical investigation.

Endosomal trafficking of two-pore K+ efflux channel TWIK2 to plasmalemma mediates NLRP3 inflammasome activation and inflammatory injury

Yamuna Krishnan, A. Malik, L. Huang, 2022, bioRxiv.

Dopaminergic Neurons During Stimulation of D2 and GABAB Receptors Voltage-Gated Calcium Channels Mediate Intracellular Calcium Increase in Weaver

D. Nelson, P. Huang, P. Hou, 2008 .

The angiocrine Rspondin3 instructs interstitial macrophage transition via metabolic–epigenetic reprogramming and resolves inflammatory injury

A. Malik, J. Rehman, L. Huang, 2020, Nature Immunology.

IL-1β Suppression of VE-cadherin transcription underlies sepsis-induced inflammatory lung injury.

A. Malik, Saroj Nepal, G. Rao, 2020, The Journal of clinical investigation.

TRP channels and the control of vascular function.

A. Malik, Anke Di, 2010, Current opinion in pharmacology.

The redox-sensitive cation channel TRPM2 modulates phagocyte ROS production and inflammation

Xiao-pei Gao, A. Malik, R. Ye, 2011, Nature Immunology.

Endothelial cell Piezo1 mediates pressure-induced lung vascular hyperpermeability via disruption of adherens junctions

A. Malik, J. Rehman, Z. Hong, 2019, Proceedings of the National Academy of Sciences.

CFTR regulates phagosome acidification in macrophages and alters bactericidal activity

V. Bindokas, F. L. Szeto, J. Tong, 2006, Nature Cell Biology.

The Interaction between Syntaxin 1A and Cystic Fibrosis Transmembrane Conductance Regulator Cl− Channels Is Mechanistically Distinct from Syntaxin 1A-SNARE Interactions*

K. Kirk, M. Quick, D. Nelson, 2003, The Journal of Biological Chemistry.

The angiocrine Rspondin3 instructs interstitial macrophage transition via metabolic–epigenetic reprogramming and resolves inflammatory injury

A. Malik, J. Rehman, L. Huang, 2020, Nature Immunology.

Cooperative Interaction of trp Melastatin Channel Transient Receptor Potential (TRPM2) With Its Splice Variant TRPM2 Short Variant Is Essential for Endothelial Cell Apoptosis

Min Zhang, Xiao-pei Gao, A. Malik, 2013, Circulation research.

Oxidant Sensing by TRPM2 Inhibits Neutrophil Migration and Mitigates Inflammation.

C. Bai, T. Jin, Yong Chen, 2016, Developmental cell.

CFTR surface expression and chloride currents are decreased by inhibitors of N-WASP and actin polymerization.

K. Kirk, D. Nelson, K. Nowotarski, 2007, Biochimica et biophysica acta.

Lysophosphatidic acid inhibits cholera toxin-induced secretory diarrhea through CFTR-dependent protein interactions

Y. Fujiwara, G. Tigyi, K. Roy, 2005, The Journal of experimental medicine.

CFTR chloride channels are regulated by a SNAP-23/syntaxin 1A complex

K. Kirk, A. Naren, D. Nelson, 2002, Proceedings of the National Academy of Sciences of the United States of America.

Dynamin regulates focal exocytosis in phagocytosing macrophages.

V. Bindokas, D. Nelson, Mary E. Brown, 2003, Molecular biology of the cell.

Quantal release of free radicals during exocytosis of phagosomes

Anjaparavanda P. Naren, K. Kirk, A. Naren, 2002, Nature Cell Biology.

Regulation of Human CLC-3 Channels by Multifunctional Ca2+/Calmodulin-dependent Protein Kinase*

M. Musch, N. C. Robinson, M. Kaetzel, 2001, The Journal of Biological Chemistry.

Mechanisms of CFTR regulation by syntaxin 1A and PKA.

K. Kirk, A. Naren, D. Nelson, 2002, Journal of cell science.

Corrigendum: The redox-sensitive cation channel TRPM2 modulates phagocyte ROS production and inflammation

A. Malik, R. Ye, S. Vogel, 2012, Nature Immunology.

5-amino-imidazole carboxamide riboside acutely potentiates glucose-stimulated insulin secretion from mouse pancreatic islets by KATP channel-dependent and -independent pathways.

L. Philipson, G. Bell, M. Magnuson, 2005, Biochemical and biophysical research communications.

IL-1β suppression of VE-cadherin transcription underlies sepsis-induced inflammatory lung injury

Wei Wu, Saroj Nepal, G. Rao, 2023, The Journal of clinical investigation.

Role of the phagosomal redox-sensitive TRP channel TRPM2 in regulating bactericidal activity of macrophages

Haixia Gong, A. Malik, Xiao‐pei Gao, 2017, Journal of Cell Science.