M. Sahu

Phosphodiesterase‐3B‐cAMP Pathway of Leptin Signalling in the Hypothalamus is Impaired During the Development of Diet‐Induced Obesity in FVB/N Mice

A. Sahu, P. Anamthathmakula, M. Sahu, 2015, Journal of neuroendocrinology.

Hypothalamic phosphatidylinositol 3-kinase pathway of leptin signaling is impaired during the development of diet-induced obesity in FVB/N mice.

A. Sahu, A. Metlakunta, M. Sahu, 2008, Endocrinology.

Hypothalamic Phosphodiesterase 3B Pathway Mediates Anorectic and Body Weight-Reducing Effects of Insulin in Male Mice

A. Sahu, P. Anamthathmakula, M. Sahu, 2016, Neuroendocrinology.

Hypothalamic PDE3B deficiency alters body weight and glucose homeostasis in mouse

A. Sahu, P. Anamthathmakula, M. Sahu, 2018, The Journal of endocrinology.

Leptin receptor expressing neurons express phosphodiesterase-3B (PDE3B) and leptin induces STAT3 activation in PDE3B neurons in the mouse hypothalamus

A. Sahu, M. Sahu, 2015, Peptides.

Neuronal suppressor of cytokine signaling-3 deficiency enhances hypothalamic leptin-dependent phosphatidylinositol 3-kinase signaling.

A. Yoshimura, D. Belsham, A. Sahu, 2011, American journal of physiology. Regulatory, integrative and comparative physiology.

Phosphatidylinositol 3‐kinase is an Upstream Regulator of the Phosphodiesterase 3B Pathway of Leptin Signalling that may not Involve Activation of Akt in the Rat Hypothalamus

A. Sahu, K. Koshinaka, M. Sahu, 2013, Journal of neuroendocrinology.

Phosphodiesterase-3B is expressed in proopiomelanocortin and neuropeptide Y neurons in the mouse hypothalamus

A. Sahu, M. Sahu, D. Litvin, 2011, Neuroscience Letters.

Neuronal suppressor of cytokine signaling-3 deficiency enhances hypothalamic leptin-dependent phosphatidylinositol 3-kinase signaling.

A. Yoshimura, D. Belsham, A. Sahu, 2011, American journal of physiology. Regulatory, integrative and comparative physiology.

Hypothalamic PDE3B deficiency alters body weight and glucose homeostasis in mouse

A. Sahu, M. Sahu, Prashanth Anamthathmakula, 2018, The Journal of endocrinology.