S. Mitarai

Early and delayed consequences of beta(2)-adrenergic receptor overexpression in mouse hearts: critical role for expression level.

G. Dorn, S. Liggett, J. Lorenz, 2000, Circulation.

Hearts : Critical Role for Expression Level-Adrenergic Receptor Overexpression in Mouse 2 β Early and Delayed Consequences of Print

G. Dorn, S. Liggett, J. Lorenz, 2000 .

Site of Action of the General Anesthetic Propofol in Muscarinic M1 Receptor-Mediated Signal Transduction

K. Sumikawa, K. Taniyama, M. Kaibara, 2003, Journal of Pharmacology and Experimental Therapeutics.

Rescue of Contractile Parameters and Myocyte Hypertrophy in Calsequestrin Overexpressing Myocardium by Phospholamban Ablation*

G. Dorn, Guangyu Wu, E. Kranias, 2001, The Journal of Biological Chemistry.

Changes in ionic currents and β-adrenergic receptor signaling in hypertrophied myocytes overexpressing Gαq

A. Yatani, T. Reed, S. Mitarai, 2000 .

Changes in ionic currents and beta-adrenergic receptor signaling in hypertrophied myocytes overexpressing G alpha(q).

A. Yatani, T. Reed, S. Mitarai, 2000, American journal of physiology. Heart and circulatory physiology.

Two distinct inactivation processes related to phosphorylation in cardiac L-type Ca(2+) channel currents.

K. Taniyama, M. Kaibara, S. Mitarai, 2000, American journal of physiology. Cell physiology.

Evidence of three clinical subgroups in patients with dual atrioventricular nodal pathways.

S. Seto, K. Yano, K. Kitano, 1997, The American journal of the medical sciences.

Involvement of Na(+)-H+ antiporter in regulation of L-type Ca2+ channel current by angiotensin II in rabbit ventricular myocytes.

M. Kameyama, M. Kaibara, S. Mitarai, 1994, Circulation research.