C. Kuhn

发表

Periprocedural Changes of NT‐proBNP Are Associated With Survival After Transcatheter Aortic Valve Implantation

S. Freitag-Wolf, N. Frey, D. Frank, 2019, Journal of the American Heart Association.

Prognostic implications of N-terminal pro-B-type natriuretic peptide in patients with normal left ventricular ejection fraction undergoing transcatheter aortic valve implantation.

N. Frey, D. Frank, H. Seoudy, 2019, International journal of cardiology.

MicroRNA miR-301a is a novel cardiac regulator of Cofilin-2

N. Frey, D. Frank, Phillip Hoppe, 2017, PloS one.

Cardiac remodeling is not modulated by overexpression of muscle LIM protein (MLP)

H. Katus, J. Backs, N. Frey, 2012, Basic Research in Cardiology.

Affixin (β-parvin) promotes cardioprotective signaling via STAT3 activation.

H. Katus, N. Frey, D. Frank, 2011, Journal of molecular and cellular cardiology.

Myomasp/LRRC39, a Heart- and Muscle-Specific Protein, Is a Novel Component of the Sarcomeric M-Band and Is Involved in Stretch Sensing

W. Rottbauer, S. Wiemann, M. Koegl, 2010, Circulation research.

Improvement of renal function after transcatheter aortic valve replacement and its impact on survival

S. Freitag-Wolf, N. Frey, T. Feldkamp, 2020, BMC Nephrology.

Incidence and impact of prosthesis–patient mismatch following transcatheter aortic valve implantation

S. Freitag-Wolf, N. Frey, D. Frank, 2018, Clinical Research in Cardiology.

Mice with cardiac-restricted overexpression of Myozap are sensitized to biomechanical stress and develop a protein-aggregate-associated cardiomyopathy.

N. Frey, C. Grund, W. Franke, 2013, Journal of molecular and cellular cardiology.

Elevated high-sensitivity troponin T levels at 1-year follow-up are associated with increased long-term mortality after TAVR

S. Freitag-Wolf, T. Puehler, N. Frey, 2020, Clinical Research in Cardiology.

Characteristics and outcomes of patients ≤ 75 years who underwent transcatheter aortic valve implantation: insights from the SOURCE 3 Registry

H. Baumgartner, T. Hovorka, M. Gilard, 2018, Clinical Research in Cardiology.

Dysbindin is a potent inducer of RhoA–SRF-mediated cardiomyocyte hypertrophy

N. Frey, D. Frank, A. Rangrez, 2013, The Journal of cell biology.

Calculated Plasma Volume Status Is Associated with Adverse Outcomes in Patients Undergoing Transcatheter Aortic Valve Implantation

T. Puehler, D. Frank, H. Seoudy, 2021, Journal of clinical medicine.

The novel cardiac z-disc protein CEFIP regulates cardiomyocyte hypertrophy by modulating calcineurin signaling

Sibylle Molt, D. Fürst, N. Frey, 2017, The Journal of Biological Chemistry.

Guidelines for the use and interpretation of assays formonitoring autophagy (3rd edition)

Sangeeta Khare, Xin Qi, Ying Wang, 2016 .

Activation of Cardiac Fibroblast Growth Factor Receptor 4 Causes Left Ventricular Hypertrophy.

A. Ullrich, M. Wolf, J. Stypmann, 2015, Cell metabolism.

Role of the sarcomeric Z-disc in the pathogenesis of cardiomyopathy.

H. Katus, N. Frey, D. Frank, 2007, Future cardiology.

The sarcomeric Z-disc: a nodal point in signalling and disease

H. Katus, N. Frey, D. Frank, 2006, Journal of Molecular Medicine.

MicroRNA-20a inhibits stress-induced cardiomyocyte apoptosis involving its novel target Egln3/PHD3.

H. Katus, N. Frey, D. Frank, 2012, Journal of molecular and cellular cardiology.

Mice lacking calsarcin-1 are sensitized to calcineurin signaling and show accelerated cardiomyopathy in response to pathological biomechanical stress

E. Olson, R. Bassel-Duby, J. Richardson, 2004, Nature Medicine.

F-Box and Leucine-Rich Repeat Protein 22 Is a Cardiac-Enriched F-Box Protein That Regulates Sarcomeric Protein Turnover and Is Essential for Maintenance of Contractile Function In Vivo

W. Rottbauer, S. Wiemann, M. Koegl, 2012, Circulation research.

Lmcd1/Dyxin, a novel Z-disc associated LIM protein, mediates cardiac hypertrophy in vitro and in vivo.

H. Katus, N. Frey, L. Füzesi, 2010, Journal of molecular and cellular cardiology.

Calsarcin-2 deficiency increases exercise capacity in mice through calcineurin/NFAT activation.

E. Olson, R. Bassel-Duby, H. Katus, 2008, The Journal of clinical investigation.

Gene Expression Pattern in Biomechanically Stretched Cardiomyocytes: Evidence for a Stretch-Specific Gene Program

Hugo A. Katus, Benedikt Brors, Norbert Frey, 2008, Hypertension.

Calsarcin-1 Protects Against Angiotensin-II–Induced Cardiac Hypertrophy

H. Katus, M. van Eickels, N. Frey, 2007, Circulation.

Dyrk1a regulates the cardiomyocyte cell cycle via D-cyclin-dependent Rb/E2f-signalling.

R. Lüllmann-Rauch, N. Frey, D. Adam, 2016, Cardiovascular research.

Myozap Deficiency Promotes Adverse Cardiac Remodeling via Differential Regulation of Mitogen-activated Protein Kinase/Serum-response Factor and β-Catenin/GSK-3β Protein Signaling*

R. Lüllmann-Rauch, N. Frey, H. Weiler, 2015, The Journal of Biological Chemistry.

DYRK1A Is a Novel Negative Regulator of Cardiomyocyte Hypertrophy*

H. Katus, N. Frey, D. Frank, 2009, The Journal of Biological Chemistry.

Adipose Tissue-Derived Soluble Fms-Like Tyrosine Kinase 1 Is an Obesity-Relevant Endogenous Paracrine Adipokine

F. Luft, S. Engeli, N. Frey, 2011, Hypertension.

The plaque protein myozap identified as a novel major component of adhering junctions in endothelia of the blood and the lymph vascular systems

N. Frey, W. Claycomb, W. Franke, 2012, Journal of cellular and molecular medicine.

Transcatheter aortic valve implantation versus surgical aortic valve replacement in patients with severe aortic stenosis: a systematic review and meta-analysis

J. Kleijnen, A. Haneya, T. Puehler, 2021, BMJ Open.

Erratum to: Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition) (Autophagy, 12, 1, 1-222, 10.1080/15548627.2015.1100356

Sara W. Bird, Erinna F. Lee, Michael D. Kim, 2016 .

Editorial :Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition). DOI: 10.1080/15548627.2015.1100356;WOS:000373595400001; 2-s2.0-85013763791&;PMID: 26799652

Lee, Duchen, Izzo, 2016 .

FYCO1 Regulates Cardiomyocyte Autophagy and Prevents Heart Failure Due to Pressure Overload In Vivo

R. Lüllmann-Rauch, N. Frey, L. Carrier, 2021, JACC. Basic to translational science.

Fibin regulates cardiomyocyte hypertrophy and causes protein-aggregate-associated cardiomyopathy in vivo

R. Lüllmann-Rauch, D. Frank, Oliver J. Müller, 2023, Frontiers in Molecular Biosciences.

Heat-shock-protein 90 protects from downregulation of HIF-1α in calcineurin-induced myocardial hypertrophy.

H. Katus, N. Frey, Christian Kuhn, 2015, Journal of molecular and cellular cardiology.

Mice with cardiac-restricted overexpression of Myozap are sensitized to biomechanical stress and develop a protein-aggregate-associated cardiomyopathy.

N. Frey, C. Grund, W. Franke, 2014, Journal of molecular and cellular cardiology.

The novel cardiac z-disc protein CEFIP regulates cardiomyocyte hypertrophy by modulating calcineurin signaling

Sibylle Molt, N. Frey, C. Rohr, 2017, The Journal of Biological Chemistry.

The cardiac z-disc protein CEFIP regulates cardiomyocyte hypertrophy by modulating calcineurin signaling

Sibylle Molt, S. Hille, Franziska Dierck, 2017 .

Adipose Tissue-Derived Soluble Fms-Like Tyrosine Kinase 1 Is an Obesity-Relevant Endogenous Paracrine Adipokine

S. Engeli, N. Frey, R. Dechend, 2011, Hypertension.