G. Krömer

发表

The multi-factorial pathogenesis of autoimmune disease.

R. Fässler, A. Ziemiecki, N. Neu, 1987, Immunology letters.

Spontaneous autoimmune thyroiditis - a bird's eye view.

R. Fässler, A. Ziemiecki, J. Möst, 1985, Immunology today.

The role of testosterone in spontaneous autoimmune thyroiditis of Obese strain (OS) chickens.

R. Fässler, G. Wick, H. Brezinschek, 1988, Journal of autoimmunity.

Genetic Basis of Spontaneous Autoimmune Thyroiditis

K. Schauenstein, K. Hála, G. Wick, 1987 .

Is Coxsackievirus B3-Induced Myocarditis in A/J Mice Mediated by an Autoimmune Response to Cardiac Myosin?

B. Ploier, N. Rose, N. Neu, 1988 .

Phenotypic analysis of skin infiltrates in comparison with peripheral blood lymphocytes, spleen cells and thymocytes in early avian scleroderma.

M. Gershwin, R. Boyd, G. Wick, 1991, Journal of autoimmunity.

Diminished glucocorticoid tonus in obese strain (OS) chickens with spontaneous autoimmune thyroiditis: increased plasma levels of a physicochemically unaltered corticosteroid-binding globulin but normal total corticosterone plasma concentration and normal glucocorticoid receptor contents in lymphoid

R. Fässler, S. Schwarz, G. Wick, 1988, Journal of steroid biochemistry.

ev22, a new endogenous avian leukosis virus locus found in chickens with spontaneous autoimmune thyroiditis

A. Ziemiecki, K. Hála, G. Wick, 2005, Archives of Virology.

A monoclonal antibody reacting with a membrane determinant expressed on activated chicken T lymphocytes

N. Neu, K. Schauenstein, K. Hála, 1986, European journal of immunology.

T cell hyperreactivity in obese strain (OS) chickens. Different mechanisms operative in spleen and peripheral blood lymphocyte activation.

K. Schauenstein, K. Hála, G. Wick, 1987, Immunobiology.

Enhanced response to Con A and production of TCGF by lymphocytes of obese strain (OS) chickens with spontaneous autoimmune thyroiditis.

K. Schauenstein, G. Wick, R. Sundick, 1985, Journal of immunology.

Avian lymphokines: an improved method for chicken IL-2 production and assay. A Con A-erythrocyte complex induces higher T cell proliferation and IL-2 production than does free mitogen.

K. Schauenstein, G. Wick, G. Krömer, 1984, Journal of immunological methods.

Elevation of corticosteroid-binding globulin in obese strain (OS) chickens: possible implications for the disturbed immunoregulation and the development of spontaneous autoimmune thyroiditis.

R. Fässler, S. Schwarz, K. Schauenstein, 1986, Journal of immunology.

Chicken-activated-T-lymphocyte-antigen (CATLA) recognized by monoclonal antibody INN-CH 16 represents the IL-2 receptor.

K. Schauenstein, K. Hála, G. Wick, 1988, Developmental and comparative immunology.

The genetic origin of murine lupus-associated autoantibodies.

R. Kofler, R. Fässler, R. Strohal, 1987, Immunology letters.

Disturbed immune-endocrine communication in autoimmune disease. Lack of corticosterone response to immune signals in obese strain chickens with spontaneous autoimmune thyroiditis.

R. Fässler, S. Schwarz, K. Schauenstein, 1987, Journal of immunology.

Analysis of lymphocytes infiltrating the thyroid gland of Obese strain chickens.

K. Schauenstein, K. Hála, G. Wick, 1985, Journal of immunology.

Mechanisms of T cell hyperreactivity in obese strain (OS) chickens with spontaneous autoimmune thyroiditis: lack in nonspecific suppression is due to a primary adherent cell defect.

R. Fässler, K. Schauenstein, G. Wick, 1987, Journal of immunology.

In vitro T cell hyperreactivity in Obese strain (OS) chickens is due to a defect in nonspecific suppressor mechanism(s).

N. Neu, K. Schauenstein, G. Wick, 1985, Journal of immunology.

In-vivo treatment with 5-azacytidine causes degeneration of central lymphatic organs and induces autoimmune disease in the chicken.

A. Csordás, K. Schauenstein, G. Wick, 1991, International journal of experimental pathology.