Yenfeng Wang

Mast cell‐mediated apoptosis of endothelial cells in vitro: A paracrine mechanism involving TNF‐α‐mediated down‐regulation of bcl‐2 expression

P. Kovanen, K. Lindstedt, N. Shiota, 2003, Journal of cellular physiology.

Activated Mast Cells Increase the Level of Endothelin-1 mRNA in Cocultured Endothelial Cells and Degrade the Secreted Peptide

M. Baumann, P. Kovanen, F. Fyhrquist, 2002, Arteriosclerosis, thrombosis, and vascular biology.

Activation of paracrine TGF‐β1 signaling upon stimulation and degranulation of rat serosal mast cells: a novel function for chymase

J. Keski‐Oja, P. Kovanen, K. Lindstedt, 2001, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.

Mast Cell Chymase Induces Smooth Muscle Cell Apoptosis by a Mechanism Involving Fibronectin Degradation and Disruption of Focal Adhesions

P. Kovanen, K. Lindstedt, M. Leskinen, 2003, Arteriosclerosis, thrombosis, and vascular biology.

Seed peptide lunasin ameliorates obesity-induced inflammation and regulates immune responses in C57BL/6J mice fed high-fat diet.

C. Hsieh, Yenfeng Wang, Mei-Jia Chou, 2020, Food and chemical toxicology : an international journal published for the British Industrial Biological Research Association.

Induction of ROS-independent JNK-activation-mediated apoptosis by a novel coumarin-derivative, DMAC, in human colon cancer cells.

Ku-Chung Chen, Mei-Hsiang Lin, Chia-Hsiung Cheng, 2014, Chemico-biological interactions.

Mast Cell Chymase Inhibits Smooth Muscle Cell Growth and Collagen Expression In Vitro: Transforming Growth Factor-&bgr;1-Dependent and -Independent Effects

P. Kovanen, K. Lindstedt, N. Shiota, 2001, Arteriosclerosis, thrombosis, and vascular biology.

Mast Cell Chymase Induces Apoptosis of Vascular Smooth Muscle Cells

D. Leszczyński, P. Kovanen, K. Lindstedt, 2000, Arteriosclerosis, thrombosis, and vascular biology.

Culture of Rat Aortic SMCs and Human Coronary Artery SMCs Aortic

D. Leszczyński, P. Kovanen, K. Lindstedt, 2001 .

Mast cell granule remnants carry LDL into smooth muscle cells of the synthetic phenotype and induce their conversion into foam cells.

P. Kovanen, K. Lindstedt, Yenfeng Wang, 1995, Arteriosclerosis, thrombosis, and vascular biology.