B. Hartmann

Cholera Toxin B Pretreatment of Macrophages and Monocytes Diminishes Their Proinflammatory Responsiveness to Lipopolysaccharide1

H. Kolb, P. Hanifi-Moghaddam, V. Burkart, 2002, The Journal of Immunology.

Nitric oxide toxicity in islet cells involves poly(ADP-ribose) polymerase activation and concomitant NAD+ depletion.

H. Kolb, A. Bürkle, K. Kröncke, 1994, Biochemical and biophysical research communications.

Metabolic activation of islet cells improves resistance against oxygen radicals or streptozocin, but not nitric oxide.

H. Kolb, V. Burkart, B. Hartmann, 1996, The Journal of clinical endocrinology and metabolism.

Islet Cell DNA Is a Target of Inflammatory Attack by Nitric Oxide

H. Kolb, K. Fehsel, A. Jalowy, 1993, Diabetes.

Fusidic acid suppresses nitric oxide toxicity in pancreatic islet cells.

H. Kolb, K. Bellmann, V. Burkart, 1994, Biochemical pharmacology.

Oral insulin for diabetes prevention in NOD mice: potentiation by enhancing Th2 cytokine expression in the gut through bacterial adjuvant

H. Kolb, R. Kleemann, K. Bellmann, 1997, Diabetologia.

Interleukin-12 gene-expression of macrophages is regulated by nitric oxide.

H. Kolb, P. Geerlings, B. Hartmann, 1996, Biochemical and biophysical research communications.

Intervention in autoimmune diabetes by targeting the gut immune system.

H. Kolb, K. Bellmann, S. Rastegar, 1997, International journal of immunopharmacology.