C. Lee

发表

2,3,7,8-Tetrachlorodibenzo-p-dioxin induction of cytochrome P450-dependent arachidonic acid metabolism in mouse liver microsomes: evidence for species-specific differences in responses.

N. Kerkvliet, B. Lawrence, A. Rifkind, 1998, Toxicology and applied pharmacology.

Identification of CYP1A5 as the CYP1A enzyme mainly responsible for uroporphyrinogen oxidation induced by AH receptor ligands in chicken liver and kidney.

P. Sinclair, N. Gorman, J. Sinclair, 1997, Drug metabolism and disposition: the biological fate of chemicals.

Effects of caffeine and theophylline on acetaminophen pharmacokinetics: P450 inhibition and activation.

S. Nelson, J. Slattery, C. Lee, 1996, The Journal of pharmacology and experimental therapeutics.

Purification and biochemical characterization of two major cytochrome P-450 isoforms induced by 2,3,7,8-tetrachlorodibenzo-p-dioxin in chick embryo liver.

J. Capdevila, A. Kanetoshi, A. Rifkind, 1994, The Journal of biological chemistry.

Liquid chromatography-mass spectrometry and liquid chromatography-NMR characterization of in vitro metabolites of a potent and irreversible peptidomimetic inhibitor of rhinovirus 3C protease.

B. Hee, B. Potts, C. Lee, 2001, Drug metabolism and disposition: the biological fate of chemicals.

Mechanism of cytochrome P450 activation by caffeine and 7,8-benzoflavone in rat liver microsomes.

S. Nelson, K. Thummel, J. Slattery, 1997, Drug metabolism and disposition: the biological fate of chemicals.

Oxidation of acetaminophen to N-acetyl-p-aminobenzoquinone imine by human CYP3A4.

S. Nelson, K. Thummel, J. Slattery, 1993, Biochemical pharmacology.

Arachidonic acid metabolism by human cytochrome P450s 2C8, 2C9, 2E1, and 1A2: regioselective oxygenation and evidence for a role for CYP2C enzymes in arachidonic acid epoxygenation in human liver microsomes.

D. Waxman, T. Chang, A. Rifkind, 1995, Archives of biochemistry and biophysics.

Characterization of the selectivity and mechanism of human cytochrome P450 inhibition by the human immunodeficiency virus-protease inhibitor nelfinavir mesylate.

S. Webber, S Webber, B V Shetty, 1998, Drug metabolism and disposition: the biological fate of chemicals.

Effect of methylxanthines on acetaminophen hepatotoxicity in various induction states.

S. Nelson, J. Slattery, T. Kalhorn, 1990, The Journal of pharmacology and experimental therapeutics.

Effect of ethanol on hepatotoxicity of acetaminophen in mice and on reactive metabolite formation by mouse and human liver microsomes.

P. Pearson, S. Nelson, K. Thummel, 1989, Toxicology and applied pharmacology.

Activation of acetaminophen-reactive metabolite formation by methylxanthines and known cytochrome P-450 activators.

S. Nelson, K. Thummel, J. Slattery, 1991, Drug metabolism and disposition: the biological fate of chemicals.