M. Coonfield

发表

Coincident thresholds of mutant protein for paralytic disease and protein aggregation caused by restrictively expressed superoxide dismutase cDNA

D. Borchelt, N. Jenkins, V. Gonzales, 2005, Neurobiology of Disease.

Histidine kinase activity of the ETR1 ethylene receptor from Arabidopsis.

G. Schaller, M. Coonfield, R. L. Gamble, 1998, Proceedings of the National Academy of Sciences of the United States of America.

Copper-binding-site-null SOD1 causes ALS in transgenic mice: aggregates of non-native SOD1 delineate a common feature.

D. Borchelt, N. Jenkins, V. Gonzales, 2003, Human molecular genetics.

Fibrillar Inclusions and Motor Neuron Degeneration in Transgenic Mice Expressing Superoxide Dismutase 1 with a Disrupted Copper-Binding Site

D. Borchelt, N. Jenkins, V. Gonzales, 2002, Neurobiology of Disease.

Notch Signaling Promotes Growth and Invasion in Uveal Melanoma

J. Handa, K. Ebrahimi, J. Harbour, 2012, Clinical Cancer Research.

Distinct Behavioral and Neuropathological Abnormalities in Transgenic Mouse Models of HD and DRPLA

D. Borchelt, D. Price, C. Ross, 2001, Neurobiology of Disease.

Environmental, pharmacological, and genetic modulation of the HD phenotype in transgenic mice

D. Borchelt, A. Savonenko, C. Ross, 2004, Experimental Neurology.

Nuclear-targeting of mutant huntingtin fragments produces Huntington's disease-like phenotypes in transgenic mice.

D. Borchelt, A. Savonenko, C. Ross, 2004, Human molecular genetics.

The Role of Two-Component Systems in Ethylene Perception

M. D. Randlett, G. Schaller, M. Coonfield, 1999 .

Coenzyme Q10 and remacemide hydrochloride ameliorate motor deficits in a Huntington's disease transgenic mouse model

D. Borchelt, C. Ross, Gabriele Schilling, 2001, Neuroscience Letters.

Notch Signaling Promotes Growth and Invasion in Uveal Melanoma

J. Handa, K. Ebrahimi, J. Harbour, 2012, Clinical Cancer Research.