David Fromholt

Coincident thresholds of mutant protein for paralytic disease and protein aggregation caused by restrictively expressed superoxide dismutase cDNA

D. Borchelt, N. Jenkins, V. Gonzales, 2005, Neurobiology of Disease.

Copper-binding-site-null SOD1 causes ALS in transgenic mice: aggregates of non-native SOD1 delineate a common feature.

D. Borchelt, N. Jenkins, V. Gonzales, 2003, Human molecular genetics.

Fibrillar Inclusions and Motor Neuron Degeneration in Transgenic Mice Expressing Superoxide Dismutase 1 with a Disrupted Copper-Binding Site

D. Borchelt, N. Jenkins, V. Gonzales, 2002, Neurobiology of Disease.

Environmental Enrichment Exacerbates Amyloid Plaque Formation in a Transgenic Mouse Model of Alzheimer Disease

D. Borchelt, V. Gonzales, J. Jankowsky, 2003, Journal of neuropathology and experimental neurology.

Environmental, pharmacological, and genetic modulation of the HD phenotype in transgenic mice

D. Borchelt, A. Savonenko, C. Ross, 2004, Experimental Neurology.

Receptor-Associated Protein (RAP) Plays a Central Role in Modulating Aβ Deposition in APP/PS1 Transgenic Mice

D. Borchelt, Nianwei Lin, Ning Li, 2008, PloS one.

Somatodendritic accumulation of misfolded SOD1-L126Z in motor neurons mediates degeneration: alphaB-crystallin modulates aggregation.

D. Borchelt, N. Jenkins, Hong Li, 2005, Human molecular genetics.