Zhihua Zhang

Identification of a Small Molecule Cyclophilin D Inhibitor for Rescuing Aβ-Mediated Mitochondrial Dysfunction.

Anuradha Roy, Jianping Li, Qinru Sun, 2016, ACS medicinal chemistry letters.

NR2B-dependent cyclophilin D translocation suppresses the recovery of synaptic transmission after oxygen-glucose deprivation.

Yongfu Wang, S. Yan, Shijun Yan, 2015, Biochimica et biophysica acta.

Role of complement C1q/C3-CR3 signaling in brain injury after experimental intracerebral hemorrhage and the effect of minocycline treatment

Yucong Peng, H. Zeng, Huaijun Chen, 2022, Frontiers in Immunology.

F1F0 ATP Synthase–Cyclophilin D Interaction Contributes to Diabetes-Induced Synaptic Dysfunction and Cognitive Decline

L. Lue, D. Walker, J. Douglas, 2016, Diabetes.

Increased neuronal PreP activity reduces Aβ accumulation, attenuates neuroinflammation and improves mitochondrial and synaptic function in Alzheimer disease's mouse model.

G. McKhann, O. Arancio, A. Sosunov, 2015, Human molecular genetics.

Increased Electron Paramagnetic Resonance Signal Correlates with Mitochondrial Dysfunction and Oxidative Stress in an Alzheimer's disease Mouse Brain.

P. Kuppusamy, A. Bratasz, J. Douglas, 2016, Journal of Alzheimer's disease : JAD.

Gain of PITRM1 peptidase in cortical neurons affords protection of mitochondrial and synaptic function in an advanced age mouse model of Alzheimer’s disease

S. Yan, S. Yan, Shijun Yan, 2021, Aging cell.