W. C. Lee

发表

Progranulin regulates neuronal outgrowth independent of Sortilin

L. Petrucelli, C. Link, J. Gass, 2012, Molecular Neurodegeneration.

Distinct brain transcriptome profiles in C9orf72-associated and sporadic ALS

Christian A. Ross, Kevin F. Bieniek, Hu Li, 2015, Nature Neuroscience.

Misregulation of human sortilin splicing leads to the generation of a nonfunctional progranulin receptor

L. Petrucelli, D. Dickson, J. Gass, 2012, Proceedings of the National Academy of Sciences.

Enzyme replacement therapy results in substantial improvements in early clinical phenotype in a mouse model of globoid cell leukodystrophy

D. Dickson, C. Eckman, M. Stallings-Mann, 2005, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.

Suppression of galactosylceramidase (GALC) expression in the twitcher mouse model of globoid cell leukodystrophy (GLD) is caused by nonsense-mediated mRNA decay (NMD)

D. Dickson, C. Eckman, M. Delucia, 2006, Neurobiology of Disease.

Molecular Characterization of Mutations That Cause Globoid Cell Leukodystrophy and Pharmacological Rescue Using Small Molecule Chemical Chaperones

C. Eckman, Christopher B Eckman, Wing C Lee, 2010, The Journal of Neuroscience.

Single‐dose intracerebroventricular administration of galactocerebrosidase improves survival in a mouse model of globoid cell leukodystrophy

D. Dickson, C. Eckman, Z. Ahmed, 2007, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.

Progranulin regulates neuronal outgrowth independent of Sortilin

L. Petrucelli, C. Link, J. Gass, 2012, Molecular Neurodegeneration.

Targeted manipulation of the sortilin–progranulin axis rescues progranulin haploinsufficiency

L. Petrucelli, I. Mackenzie, Jeannie Chew, 2013, Human molecular genetics.

The dual functions of the extreme N-terminus of TDP-43 in regulating its biological activity and inclusion formation

Leonard Petrucelli, L. Petrucelli, T. Caulfield, 2013, Human molecular genetics.

Deletion of the Ubiquitin Ligase CHIP Leads to the Accumulation, But Not the Aggregation, of Both Endogenous Phospho- and Caspase-3-Cleaved Tau Species

Wen-Lang Lin, Guy A. Caldwell, Dennis W. Dickson, 2006, The Journal of Neuroscience.

Antisense transcripts of the expanded C9ORF72 hexanucleotide repeat form nuclear RNA foci and undergo repeat-associated non-ATG translation in c9FTD/ALS

Kevin F. Bieniek, L. Petrucelli, D. Dickson, 2013, Acta Neuropathologica.

Molecular cloning and expression analysis of human glycogen synthase kinase-3α promoter

P. Shaw, K. Lau, Kai-Fai Lee, 2000 .

Pharmacologic reductions of total tau levels; implications for the role of microtubule dynamics in regulating tau expression

L. Petrucelli, M. Hutton, C. Eckman, 2006, Molecular Neurodegeneration.

HSP induction mediates selective clearance of tau phosphorylated at proline‐directed Ser/Thr sites but not KXGS (MARK) sites

L. Petrucelli, M. Hutton, C. Eckman, 2006, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.