Yoko Machida

发表

Identification of ubiquitin‐interacting proteins in purified polyglutamine aggregates

Y. Kuroiwa, N. Nukina, H. Doi, 2004, FEBS letters.

A novel therapeutic strategy for polyglutamine diseases by stabilizing aggregation-prone proteins with small molecules

N. Nukina, Motomasa Tanaka, Yoko Machida, 2005, Journal of Molecular Medicine.

Trehalose alleviates polyglutamine-mediated pathology in a mouse model of Huntington disease

N. Nukina, N. Jana, H. Doi, 2004, Nature Medicine.

RNA-binding Protein TLS Is a Major Nuclear Aggregate-interacting Protein in Huntingtin Exon 1 with Expanded Polyglutamine-expressing Cells*

Y. Kuroiwa, N. Nukina, H. Shimizu, 2008, Journal of Biological Chemistry.

rAAV-mediated shRNA ameliorated neuropathology in Huntington disease model mouse.

N. Nukina, K. Ozawa, F. Oyama, 2006, Biochemical and biophysical research communications.

Decreased expression of hypothalamic neuropeptides in Huntington disease transgenic mice with expanded polyglutamine‐EGFP fluorescent aggregates

N. Nukina, N. Jana, K. Oyanagi, 2005, Journal of neurochemistry.

Blocking acid-sensing ion channel 1 alleviates Huntington's disease pathology via an ubiquitin-proteasome system-dependent mechanism.

T. Knöpfel, N. Nukina, A. Goswami, 2008, Human molecular genetics.

Sodium channel β4 subunit: down‐regulation and possible involvement in neuritic degeneration in Huntington's disease transgenic mice

Takashi Sakurai, Naoaki Sakamoto, Nobuyuki Nukina, 2006, Journal of neurochemistry.

Expansion of Polyglutamine Induces the Formation of Quasi-aggregate in the Early Stage of Protein Fibrillization*

T. Hashikawa, N. Nukina, Y. Nishikawa, 2003, Journal of Biological Chemistry.

The effects of aggregation-inducing motifs on amyloid formation of model proteins related to neurodegenerative diseases.

T. Hashikawa, N. Nukina, I. Morishima, 2002, Biochemistry.