Nícolas Manzke Glanzel

Bioenergetics dysfunction, mitochondrial permeability transition pore opening and lipid peroxidation induced by hydrogen sulfide as relevant pathomechanisms underlying the neurological dysfunction characteristic of ethylmalonic encephalopathy.

G. Leipnitz, M. Wajner, A. U. Amaral, 2017, Biochimica et biophysica acta. Molecular basis of disease.

Evidence that thiol group modification and reactive oxygen species are involved in hydrogen sulfide-induced mitochondrial permeability transition pore opening in rat cerebellum.

G. Leipnitz, M. Wajner, A. U. Amaral, 2019, Mitochondrion.

Sulfite Impairs Bioenergetics and Redox Status in Neonatal Rat Brain: Insights into the Early Neuropathophysiology of Isolated Sulfite Oxidase and Molybdenum Cofactor Deficiencies

G. Leipnitz, M. Wajner, A. Wyse, 2023, Cellular and Molecular Neurobiology.