J. Whitlock

发表

2,3,7,8‐Tetrachlorodibenzo‐p‐dioxin receptors regulate transcription of the cytochrome P1‐450 gene

Phillip B. C. Jones, L. Durrin, D. Galeazzi, 1987, Journal of cellular biochemistry.

Heterogeneity in the Rate of Benzo[a]pyrene Metabolism in Single Cells: Quantitation Using Flow Cytometry

A. Miller, J. Whitlock, 1982, Molecular and cellular biology.

Xenobiotic-inducible Transcription of Cytochrome P450 Genes (*)

M. Denison, J. Whitlock, 1995, The Journal of Biological Chemistry.

ARYL HYDROCARBON HYDROXYLASE AND BENZO[A] PYRENE METABOLISM IN RODENT LIVER AND HUMAN CELLS

J. Whitlock, H. Gelboin, J. Whitlock, 1980 .

Hypoxia-inducible Mammalian Gene Expression Analyzed in Vivo at a TATA-driven Promoter and at an Initiator-driven Promoter*

S. Okino, J. P. Whitlock, C. Chichester, 1998, The Journal of Biological Chemistry.

Induction of Phosphoglycerate Kinase 1 Gene Expression by Hypoxia

Hui Li, H. Ko, J. Whitlock, 1996, The Journal of Biological Chemistry.

Regulation of GLUT1 Gene Transcription by the Serine/Threonine Kinase Akt1*

Richard A. Roth, A. Barthel, R. Roth, 1999, The Journal of Biological Chemistry.

Down-regulation of Major Histocompatibility ComplexQ1 b Gene Expression by 2,3,7,8-Tetrachlorodibenzo-p-dioxin*

J. Whitlock, Q. Ma, L. Dong, 1997, The Journal of Biological Chemistry.

A Novel Cytoplasmic Protein That Interacts with the Ah Receptor, Contains Tetratricopeptide Repeat Motifs, and Augments the Transcriptional Response to 2,3,7,8-Tetrachlorodibenzo-p-dioxin*

J. P. Whitlock, Qiang Ma, J. Whitlock, 1997, The Journal of Biological Chemistry.

DNA Binding by the Heterodimeric Ah Receptor

J. P. Whitlock, Liqun Dong, Qiang Ma, 1996, The Journal of Biological Chemistry.

Analysis of CYP1A1 promoter function by transcription in vitro

Keith Jones, L. Wen, J. Whitlock, 1991, Molecular carcinogenesis.

Activation of transcription as a general mechanism of 2,3,7,8‐tetrachlorodibenzo‐p‐dioxin action

J. Whitlock, K. Jones, J. Fisher, 1989, Molecular carcinogenesis.

Activation of cytochrome P450IA1 gene expression by 2, 3, 7, 8‐tetrachlorodibenzo‐p‐dioxin in wild‐type and high‐activity variant mouse hepatoma cells

Keith Jones, J. Whitlock, Margaret A. Hirst, 1989, Molecular carcinogenesis.

Dioxin-inducible Transactivation in a Chromosomal Setting

L. Jones, J. Whitlock, 2001, The Journal of Biological Chemistry.

A Novel Response to Dioxin

J. Whitlock, L. Dong, Liqun Dong, 1998, The Journal of Biological Chemistry.

Transcriptional Activation by the Mouse Ah Receptor.

Liqun Dong, Qiang Ma, J. Whitlock, 1995, The Journal of Biological Chemistry.

Dioxin-induced CYP1A1 transcription in vivo: the aromatic hydrocarbon receptor mediates transactivation, enhancer-promoter communication, and changes in chromatin structure

J. Whitlock, S. Okino, Q. Ma, 1996, Molecular and cellular biology.

Inducible, receptor-dependent protein-DNA interactions at a dioxin-responsive transcriptional enhancer.

J. Whitlock, M. Denison, J. Fisher, 1988, Proceedings of the National Academy of Sciences of the United States of America.

The DNA recognition site for the dioxin-Ah receptor complex. Nucleotide sequence and functional analysis.

J. Whitlock, M. Denison, J. Fisher, 1988, The Journal of biological chemistry.

Transactivation domains facilitate promoter occupancy for the dioxin-inducible CYP1A1 gene in vivo

J. Whitlock, S. Okino, Q. Ma, 1997, Molecular and cellular biology.

Mechanistic aspects of dioxin action.

J. Whitlock, J. Whitlock, 1993, Chemical research in toxicology.

The control of cytochrome P-450 gene expression by dioxin.

J. Whitlock, J. Whitlock, 1989, Trends in pharmacological sciences.

The aromatic hydrocarbon receptor modulates the Hepa 1c1c7 cell cycle and differentiated state independently of dioxin

J. Whitlock, Q. Ma, Qiang Ma, 1996, Molecular and cellular biology.

Protein-DNA interactions at recognition sites for the dioxin-Ah receptor complex.

J. Whitlock, M. Denison, J. M. Fisher, 1989, The Journal of biological chemistry.

Use of cDNA microarrays to analyze dioxin-induced changes in human liver gene expression.

P. Brown, K. Hayashibara, F. Frueh, 2001, Toxicology letters.

The aromatic hydrocarbon receptor, transcription, and endocrine aspects of dioxin action.

J. Whitlock, S. Okino, J. Whitlock, 2000, Vitamins and hormones.

Organization and function of a dioxin-responsive enhancer.

L. Wu, J. Whitlock, M. Denison, 1990, The Journal of biological chemistry.

2,3,7,8-Tetrachlorodibenzo-p-dioxin-inducible, Ah receptor-mediated bending of enhancer DNA.

J. Whitlock, C. Elferink, J. Whitlock, 1990, The Journal of biological chemistry.

Mechanism of dioxin action: Ah receptor-mediated increase in promoter accessibility in vivo.

L. Wu, J. Whitlock, J. Whitlock, 1992, Proceedings of the National Academy of Sciences of the United States of America.