C. A. Hewson

Preclinical Evaluation of an Inhibitor of Cytosolic Phospholipase A2α for the Treatment of Asthma

M. Cazzola, J. D. Clark, M. Yeadon, 2012, Journal of Pharmacology and Experimental Therapeutics.

RHINOVIRUS INDUCES MUC5AC IN A HUMAN INFECTION MODEL, & IN VITRO VIA NF-κB & EGFR PATHWAYS

S. Johnston, Jie Zhu, T. Kebadze, 2010 .

Rhinovirus induces MUC5AC in a human infection model and in vitro via NF-&kgr;B and EGFR pathways

S. Johnston, P. Jeffery, S. Johnston, 2010, European Respiratory Journal.

Design and synthesis of long acting inhaled corticosteroids for the treatment of asthma.

David S. Millan, M. Yeadon, P. Glossop, 2011, Bioorganic & medicinal chemistry letters.

A New Series of Orally Bioavailable Chemokine Receptor 9 (CCR9) Antagonists; Possible Agents for the Treatment of Inflammatory Bowel Disease.

V. Viswanadhan, B. Kulkarni, J. Aiyar, 2016, Journal of medicinal chemistry.

PMA induces the MUC5AC respiratory mucin in human bronchial epithelial cells, via PKC, EGF/TGF-alpha, Ras/Raf, MEK, ERK and Sp1-dependent mechanisms.

S. Johnston, M. Edbrooke, C. A. Hewson, 2004, Journal of molecular biology.

Protein kinase R, IκB kinase-β and NF-κB are required for human rhinovirus induced pro-inflammatory cytokine production in bronchial epithelial cells

S. Johnston, N. Mukaida, V. Laza-Stanca, 2007 .

Toll-Like Receptor 3 Is Induced by and Mediates Antiviral Activity against Rhinovirus Infection of Human Bronchial Epithelial Cells

S. Johnston, V. Laza-Stanca, M. Edwards, 2005, Journal of Virology.

Protein kinase R, IkappaB kinase-beta and NF-kappaB are required for human rhinovirus induced pro-inflammatory cytokine production in bronchial epithelial cells.

S. Johnston, N. Mukaida, V. Laza-Stanca, 2007, Molecular immunology.

A differential role for ceramide kinase in antigen/FcɛRI‐mediated mast cell activation and function

J. Watson, C. A. Hewson, W. Liu, 2011, Clinical and experimental allergy : journal of the British Society for Allergy and Clinical Immunology.