Benjamin L. Cornett

CB1 Knockout Mice Unveil Sustained CB2-Mediated Antiallodynic Effects of the Mixed CB1/CB2 Agonist CP55,940 in a Mouse Model of Paclitaxel-Induced Neuropathic Pain

K. Mackie, A. Hohmann, Liting Deng, 2015, Molecular Pharmacology.

Chronic Cannabinoid Receptor 2 Activation Reverses Paclitaxel Neuropathy Without Tolerance or Cannabinoid Receptor 1–Dependent Withdrawal

K. Mackie, A. Hohmann, A. Makriyannis, 2015, Biological Psychiatry.

A pro-nociceptive phenotype unmasked in mice lacking fatty-acid amide hydrolase

K. Mackie, H. Bradshaw, A. Hohmann, 2016, Molecular pain.

Chronic cannabinoid CB 2 activation reverses paclitaxel neuropathy without tolerance or CB 1 -dependent withdrawal

A. Hohmann, A. Makriyannis, J. Guindon, 2015 .

Broad impact of deleting endogenous cannabinoid hydrolyzing enzymes and the CB1 cannabinoid receptor on the endogenous cannabinoid-related lipidome in eight regions of the mouse brain.

K. Mackie, H. Bradshaw, E. Leishman, 2016, Pharmacological research.

CB1 Knockout Mice Unveil Sustained CB2-Mediated Antiallodynic Effects of the Mixed CB1/CB2 Agonist CP55,940 in a Mouse Model of Paclitaxel-Induced Neuropathic Pain

K. Mackie, A. Hohmann, Liting Deng, 2015, Molecular Pharmacology.