P. Sokol

发表

Pentachlorobutadienyl-L-cysteine (PCBC) toxicity: the importance of mitochondrial dysfunction.

E. Lock, R. Schnellmann, T. Steffens, 1991, Journal of biochemical toxicology.

The neurotoxins 1-methyl-4-phenylpyridinium and 1-methyl-4-phenyl-1,2,3,6- tetrahydropyridine are substrates for the organic cation transporter in renal brush border membrane vesicles.

C. Ross, P. Holohan, P. Sokol, 1987, The Journal of pharmacology and experimental therapeutics.

Sulfhydryl groups are essential for organic anion exchange in canine renal brush-border membranes.

C. Ross, P. Holohan, P. Sokol, 1986, Biochimica et biophysica acta.

Mechanism of organic cation transport in rabbit renal basolateral membrane vesicles.

T. Mckinney, P. Sokol, 1990, The American journal of physiology.

Effect of endogenous and exogenous polyamines on organic cation transport in rabbit renal plasma membrane vesicles.

S. B. Gates, P. Sokol, 1990, The Journal of pharmacology and experimental therapeutics.

Sulfhydryl groups are essential for organic cation exchange in rabbit renal basolateral membrane vesicles.

T. Mckinney, W. B. Zimmerman, P. Sokol, 1991, The Journal of biological chemistry.

Mechanism of ochratoxin A transport in kidney.

C. Ross, P. Holohan, P. Sokol, 1988, The Journal of pharmacology and experimental therapeutics.

Effect of 3'-azido-3'-deoxythymidine (AZT) on organic ion transport in rat renal brush border membrane vesicles.

S. Hall, P. Sokol, D. Griffiths, 1992, The Journal of pharmacology and experimental therapeutics.

Interaction of 3'-azido-3'-deoxythymidine with organic ion transport in rat renal basolateral membrane vesicles.

S. Hall, P. Sokol, D. Griffiths, 1991, The Journal of pharmacology and experimental therapeutics.

Mechanism of vancomycin transport in the kidney: studies in rabbit renal brush border and basolateral membrane vesicles.

P. Sokol, 1991, The Journal of pharmacology and experimental therapeutics.

Gentamicin and verapamil compete for a common transport mechanism in renal brush border membrane vesicles.

C. Ross, P. Holohan, P. Sokol, 1989, The Journal of pharmacology and experimental therapeutics.

Arginyl and histidyl groups are essential for organic anion exchange in renal brush-border membrane vesicles.

C. Ross, P. Holohan, P. Sokol, 1988, The Journal of biological chemistry.

Proton-coupled organic cation transport in renal brush-border membrane vesicles.

C. Ross, S. Grassl, P. Holohan, 1988, Biochimica et biophysica acta.

Cyclosporin A and vehicle toxicity in primary cultures of rabbit renal proximal tubule cells.

C. Ross, D. Jones, R. Coulson, 1990, The American journal of physiology.

Electroneutral transport of organic cations in canine renal brush border membrane vesicles (BBMV).

C. Ross, P. Holohan, P. Sokol, 1985, The Journal of pharmacology and experimental therapeutics.

Gentamicin-induced increases in cytosolic calcium in pig kidney cells (LLC-PK1).

C. Ross, D. Laska, R. Coulson, 1988, The Journal of pharmacology and experimental therapeutics.

Photoaffinity labeling of the organic cation/H+ exchanger in renal brush border membrane vesicles.

K. White, J. Rebbeor, P. Holohan, 1992, The Journal of biological chemistry.

Effect of DQ-2556, a new cephalosporin, on organic ion transport in renal plasma membrane vesicles from the dog, rabbit and rat.

P. Sokol, 1990, The Journal of pharmacology and experimental therapeutics.

N,N'-dicyclohexylcarbodiimide inactivates organic cation transport in renal brush border membranes.

C. Ross, P. Holohan, P. Sokol, 1987, Journal of Pharmacology and Experimental Therapeutics.

Essential disulfide and sulfhydryl groups for organic cation transport in renal brush-border membranes.

C. Ross, P. Holohan, P. Sokol, 1986, The Journal of biological chemistry.

Acridine orange transport in canine renal brush-border membrane vesicles.

C. Ross, P. Holohan, P. Sokol, 1990, Biochemical pharmacology.

Mechanism of putrescine transport in human pulmonary artery endothelial cells.

K. Longenecker, W. Martin, D. Kachel, 1993, Journal of Pharmacology and Experimental Therapeutics.