H. Jaeschke

发表

Mechanisms of hypoxic cell injury. Summary of the symposium presented at the 1990 annual meeting of the Society of Toxicology.

J. Lemasters, J. Kehrer, J. Farber, 1990, Toxicology and applied pharmacology.

Intercellular adhesion molecule 1 (ICAM‐1) expression and its role in neutrophil‐induced ischemia‐reperfusion injury in rat liver

C. Smith, A. Manning, A. Farhood, 1995, Journal of leukocyte biology.

REVIEW Mechanisms of Immune-Mediated Liver Injury

D. Adams, S. Ramaiah, J. Uetrecht, 2010 .

Are cultured liver cells the right tool to investigate mechanisms of liver disease or hepatotoxicity?

H. Jaeschke, 2003, Hepatology.

A non-human primate model of acute liver failure suitable for testing liver support systems

S. Nyberg, J. Platt, A. Soto-Gutiérrez, 2022, Frontiers in Medicine.

Mechanism of cell death during warm hepatic ischemia‐reperfusion in rats: Apoptosis or necrosis?

A. Farhood, T. Bucci, J. Gujral, 2001, Hepatology.

ACTIVATION OF KUPFFER CELLS AND NEUTROPHILS FOR REACTIVE OXYGEN FORMATION IS RESPONSIBLE FOR ENDOTOXIN-ENHANCED LIVER INJURY AFTER HEPATIC ISCHEMIA

C. Smith, Peitan Liu, M. Fisher, 1995, Shock.

Complement and tumor necrosis factor‐α contribute to Mac‐1 (CD11b/CD18) up‐regulation and systemic neutrophil activation during endotoxemia in vivo

C. Smith, H. Jaeschke, A. Farhood, 1994, Journal of leukocyte biology.

Kupffer cell–induced oxidant stress during hepatic ischemia‐reperfusion: Does the controversy continue?

H. Jaeschke, 1999, Hepatology.

Parenchymal cell apoptosis as a signal for sinusoidal sequestration and transendothelial migration of neutrophils in murine models of endotoxin and fas‐antibody–induced liver injury

H. Jaeschke, M. Fisher, A. Farhood, 1998, Hepatology.

INHIBITION OF NITRIC OXIDE SYNTHESIS AGGRAVATES REPERFUSION INJURY AFTER HEPATIC ISCHEMIA AND ENDOTOXEMIA

A. Farhood, W. Mathews, Ying Wang, 1995, Shock.

Antineutrophil monoclonal antibody (1F12) alters superoxide anion release by neutrophils and Kupffer cells

C. Smith, H. Jaeschke, J. Spitzer, 1994, Journal of leukocyte biology.

Contribution of complement‐stimulated hepatic macrophages and neutrophils to endotoxin‐induced liver injury in rats

C. Smith, A. Farhood, H. Jaeschke, 1994, Hepatology.

SEQUESTRATION OF NEUTROPHILS IN THE HEPATIC VASCULATURE DURING ENDOTOXEMIA IS INDEPENDENT OF β2 INTEGRINS AND INTERCELLULAR ADHESION MOLECULE‐1

C. Smith, M. Fisher, A. Farhood, 1996, Shock.

Monoclonal antibody against the CD18 adhesion molecule stimulates glucose uptake by the liver and hepatic nonparenchymal cells

C. Smith, H. Jaeschke, J. Spitzer, 1993, Hepatology.

Activation of caspase 3 (CPP32)-like proteases is essential for TNF-alpha-induced hepatic parenchymal cell apoptosis and neutrophil-mediated necrosis in a murine endotoxin shock model.

H. Jaeschke, D. Jones, M. Fisher, 1998, Journal of immunology.

Neutrophil and Kupffer cell-induced oxidant stress and ischemia-reperfusion injury in rat liver.

H. Jaeschke, A. Farhood, H. Jaeschke, 1991, The American journal of physiology.

Reactive oxygen and ischemia/reperfusion injury of the liver.

H. Jaeschke, H. Jaeschke, 1991, Chemico-biological interactions.

Endotoxin-induced activation of the nuclear transcription factor kappa B and expression of E-selectin messenger RNA in hepatocytes, Kupffer cells, and endothelial cells in vivo.

H. Jaeschke, A. Manning, N. Essani, 1996, Journal of immunology.

Superoxide generation by Kupffer cells and priming of neutrophils during reperfusion after hepatic ischemia.

H. Jaeschke, J. Spitzer, Z. Spolarics, 1991, Free radical research communications.

Mechanisms of reperfusion injury after warm ischemia of the liver.

H. Jaeschke, H. Jaeschke, 1998, Journal of hepato-biliary-pancreatic surgery.

Protection against Fas receptor-mediated apoptosis in hepatocytes and nonparenchymal cells by a caspase-8 inhibitor in vivo: evidence for a postmitochondrial processing of caspase-8.

S. Vonderfecht, M. Bajt, J. A. Lawson, 2000, Toxicological sciences : an official journal of the Society of Toxicology.

Preservation injury: mechanisms, prevention and consequences.

H. Jaeschke, H. Jaeschke, 1996, Journal of hepatology.

Inhibition of Fas receptor (CD95)-induced hepatic caspase activation and apoptosis by acetaminophen in mice.

H. Jaeschke, M. Fisher, A. Farhood, 1999, Toxicology and applied pharmacology.

The hepatic inflammatory response after acetaminophen overdose: role of neutrophils.

A. Farhood, M. Bajt, J. A. Lawson, 2000, Toxicological sciences : an official journal of the Society of Toxicology.

Effects of CXC chemokines on neutrophil activation and sequestration in hepatic vasculature.

A. Farhood, M. Bajt, H. Jaeschke, 2001, American journal of physiology. Gastrointestinal and liver physiology.

Vascular and hepatocellular peroxynitrite formation during acetaminophen toxicity: role of mitochondrial oxidant stress.

J. Hinson, M. Bajt, H. Jaeschke, 2001, Toxicological sciences : an official journal of the Society of Toxicology.

Lipid peroxidation as molecular mechanism of liver cell injury during reperfusion after ischemia.

H. Jaeschke, M. Fisher, W. Mathews, 1994, Free radical biology & medicine.

Beneficial effects of extracellular glutathione against endotoxin-induced liver injury during ischemia and reperfusion.

C. Smith, H. Jaeschke, Peitan Liu, 1994, Circulatory shock.

Reactive oxygen species during ischemia-reflow injury in isolated perfused rat liver.

C. V. Smith, H. Jaeschke, J. Mitchell, 1988, The Journal of clinical investigation.

Role of reactive oxygen species for hepatocellular injury and heme oxygenase-1 gene expression after hemorrhage and resuscitation.

H. Jaeschke, M. Silomon, M. Bauer, 1999, Shock.

Role of nitric oxide in the oxidant stress during ischemia/reperfusion injury of the liver.

H. Jaeschke, A. Farhood, V. Schini, 1992, Life sciences.

Monoclonal antibody against the CD18 adhesion molecule stimulates glucose uptake by the liver and hepatic nonparenchymal cells

J. Spitzer, Z. Spolarics, A. Bautista, 1993, Hepatology.

Mechanisms of inflammatory liver injury: adhesion molecules and cytotoxicity of neutrophils.

C. Smith, H. Jaeschke, M. Clemens, 1996, Toxicology and applied pharmacology.

Release of soluble intercellular adhesion molecule 1 into bile and serum in murine endotoxin shock

C. Smith, H. Jaeschke, N. Essani, 1996, Hepatology.

Protection against TNF-induced liver parenchymal cell apoptosis during endotoxemia by a novel caspase inhibitor in mice.

H. Jaeschke, A. Farhood, B. Tseng, 2000, Toxicology and applied pharmacology.

Complement activates Kupffer cells and neutrophils during reperfusion after hepatic ischemia.

H. Jaeschke, J. Spitzer, A. Farhood, 1993, The American journal of physiology.

Pathophysiological consequences of enhanced intracellular superoxide formation in isolated perfused rat liver.

H. Jaeschke, A. E. Benzick, H. Jaeschke, 1992, Chemico-biological interactions.

Enhanced sinusoidal glutathione efflux during endotoxin-induced oxidant stress in vivo.

H. Jaeschke, H. Jaeschke, 1992, The American journal of physiology.

Differential protection with inhibitors of caspase-8 and caspase-3 in murine models of tumor necrosis factor and Fas receptor-mediated hepatocellular apoptosis.

H. Jaeschke, S. Vonderfecht, M. Bajt, 2001, Toxicology and applied pharmacology.

Contribution of complement‐stimulated hepatic macrophages and neutrophils to endotoxin‐induced liver injury in rats

A. Farhood, H. Jaeschke, C. Smith, 1994, Hepatology.

Transcriptional activation of vascular cell adhesion molecule-1 gene in vivo and its role in the pathophysiology of neutrophil-induced liver injury in murine endotoxin shock.

N. Essani, A. Farhood, S. Vonderfecht, 1997, Journal of immunology.

Neutrophil margination and extravasation in sinusoids and venules of liver during endotoxin-induced injury.

C. J. Dunn, H. Jaeschke, N. Essani, 1997, The American journal of physiology.

Prevention of Kupffer cell-induced oxidant injury in rat liver by atrial natriuretic peptide

A. Vollmar, H. Jaeschke, A. Gerbes, 1999 .

EFFECT OF ENDOTOXIN‐ENHANCED HEPATIC REPERFUSION INJURY ON ENDOTHELIUM‐DEPENDENT RELAXATION IN RAT AORTA

W. Mathews, R. Laskey, Ying Wang, 1996, Shock.

Neutrophil-induced lung damage after hepatic ischemia and endotoxemia.

H. Jaeschke, Peitan Liu, P. Liu, 1996, Free radical biology & medicine.